Categories
Overall Health

How Do I Check My Blood Sugar Level?

Follow your doctor’s advice and the instructions that come with the glucose meter. In general, you will follow the steps below. Different meters work differently, so be sure to check with your doctor for advice specifically for you.

  1. Wash your hands and dry them well before doing the test.
  2. Use an alcohol pad to clean the area that you’re going to prick. For most glucose meters, you will prick your fingertip. However, with some meters, you can also use your forearm, thigh, or the fleshy part of your hand. Ask your doctor what area you should use with your meter.
  3. Prick yourself with a sterile lancet to get a drop of blood. (If you prick your fingertip, it may be easier and less painful to prick it on one side, not on the pad.)
  4. Place the drop of blood on the test strip.
  5. Follow the instructions for inserting the test strip into your glucose meter.
  6. The meter will give you a number for your blood sugar level.

Tips on blood sugar testing

  1. Pay attention to expiration dates for test strips.
  2. Use a big enough drop of blood.
  3. Be sure your meter is set correctly.
  4. Keep your meter clean.
  5. Check the batteries of your meter.
  6. Follow the instructions for the test carefully.
  7. Write down the results and show them to your doctor.

How often should I check my blood sugar level?

Check your blood sugar as often as your doctor suggests. You’ll probably need to do it more often at first. You’ll also check it more often when you feel sick or stressed, when you’re changing your medicine or if you’re pregnant. People taking insulin may need to check their levels more often.

Keep track of your blood sugar levels by writing them down. You can also keep track of what you’ve eaten and how active you’ve been during the day. This will help you see how food and exercise affect your blood sugar level.

What should my blood sugar level be?

Talk with your doctor about what is a healthy blood sugar level range for you. A level of 80 to 120 before meals is often a good goal, but not everyone who has diabetes can get their blood sugar level this low.

Be sure to talk with your doctor about what to do if your blood sugar level isn’t within the range that’s best for you.

How does food affect my blood sugar level?

Anytime you eat, you put sugar in your blood. Eating the right way can help control your blood sugar level.

You need to learn how what you eat affects your blood sugar level, how you feel, and your overall health. As a general rule, just following a healthy diet is wise. Your doctor may suggest you meet with a dietitian who can teach you how to make healthier food choices.

Categories
Senior Health

Erectile Dysfunction In Aging Male

Composite case studies have been selected to identify and illustrate the dysfunctional characteristics of the male aging process.

Both Mr. and Mrs. A were 66 and 62 years of age when referred to the Foundation for sexual inadequacy. They had been married 39 years and had three children, the youngest of whom was 23 years of age. All children were married and living outside the home.

They had maintained reasonably effective sexual interchange during their marriage.

Mr. A had no difficulty with erection, reasonable ejaculatory control, and, aside from two occasions of prostitute exposure, had been fully committed to the marriage. Mrs. A occasionally orgasmic during intercourse and regularly orgasmic during her occasional masturbatory experiences had continued regularity of coital exposure with her husband until five years before referral for therapy.

Mr. A had recently retired from a major manufacturing concern. He had been relatively successful in his work and there were no specific financial problems facing man and wife during their declining years.

Both members of the marital unit had enjoyed good health throughout the marriage. At age 61, he had taken his wife abroad on a vacation trip which entailed many sightseeing trips with a different city on the agenda almost every day.

They were chronically tired during the exhausting trip, but because they were on vacation and away from home there was a definite increase over the established frequency of coital connections. Mr. A noted for the first time slowed erective attainment.

Regardless of his level of sexual interest or the depth of his wife’s commitment to the specific sexual experience, it took him progressively longer to attain a full erection. With each sexual exposure his concern for the delay in erective security increased until finally, just before termination of the vacation trip, he failed for the first time to achieve an erection quality sufficient for vaginal penetration.

When the coital opportunity first developed after return home, erection was attained, but again it was quite slow in development. The next two opportunities were only partially successful from an erective point of view, and thereafter he was secondarily impotent.

After several months they consulted their physician and were assured that this loss of erective power comes to all men as they age and that there was nothing to be done. Loath to accept the verdict, they tried on several occasions to force an erection with no success. Mr. A was seriously depressed for several months but recovered without apparent incident.

Approximately 18 months after the vacation trip, the couple had accepted their “fate.” The impotence was acknowledged to be a natural result of the aging process. This resigned attitude lasted approximately four years.

Although initially the marital unit and their physician had fallen into the socio-cultural trap of accepting the concept of sexual inadequacy as an aging phenomenon, the more Mr. and Mrs. A considered their dysfunction the less willing they were to accept the blanket concept that lack of erective security was purely the result of the aging process.

They reasoned that they were in good health, had no basic concerns as a marital unit, and took good care of themselves physically. Therefore, why was this dysfunction to be expected simply because some of their friends reportedly had accepted the loss of male erective prowess as a natural occurrence?

Each partner underwent a thorough medical checkup and sought several authoritative opinions, refusing to accept the concept of the irreversibility of their sexual distress. Finally, approximately five years after the onset of a full degree of secondary impotence, they were referred for treatment.

Sexual functioning was reconstituted for this marital unit within the first week after they arrived at the Foundation and as soon as they could absorb and accept the basic material directed toward the variation in the physiological functioning of the aging male.

No longer were they concerned with the delay in erective attainment; there were no more attempts to will, force, or strain to accomplish erection under assumed pressures of performance.

In short:
They needed only the security of the knowledge that the response pattern which initially had raised the basic fear of dysfunction was a perfectly natural result of the involutional process.

When they could accept the fact that it naturally took longer for an older man to achieve an erection, particularly if he were tired or distracted, the basis for their own sexual inadequacy disappeared.

Some six years after termination of the acute phase of therapy, this couple, now in the early seventies and late sixties, continue coital connection once or twice a week.

The husband has learned to ejaculate on his own demand schedule, and neither partner attempts a rapid return to sexual function after a mutually satisfactory sexual episode.

Husband and wife B

The husband, age 62, and his wife, age 63, were referred to the Foundation. They had two children, both of whom were married and lived out of the home.

Their sexual dysfunction had begun when the husband was 57 years old. He had noted some delay in attaining erection and marked reduction in ejaculatory volume and was particularly concerned with the fact that the ejaculatory experience was one of the mere dribbling of seminal fluid from the external urethral meatus, under obviously reduced pressure.

All these involutional signs and symptoms developed within approximately a year after he had noticed some delay in onset of erection attainment.

The more he worried about his symptoms the more frequent the occasions of impotence.

Mrs. B was completely convinced that this pattern of sexual involution was true to be expected as part of the aging process.

Rather than distress her husband, she suggested that they use separate bedrooms.

She changed from a pattern of free and easy exchange of sexual demand to one of availability for coital connection only at her husband’s expression of interest. To resolve her own sexual tensions, she masturbated about once every ten days to two weeks without her husband’s knowledge.

Finally, Mr. B developed severe prostatic spasm with ejaculation during approximately half the increasingly rare occasions when there was sufficient erective security to establish a coital connection. It was the persistence of this symptom of pain that first brought medical consultation and ultimately referral for treatment.

In the evaluation of this man during the physical examination, there was marked muscular weakness noted, a history of easy fatigability, and increasing lassitude in physical expression.

Mr. B also had been distressed in the last two to three years before referral to therapy with distinct memory loss for recent events. He described the loss of work effectiveness for approximately the same length of time.

With these overt symptoms suggestive of steroid starvation, testosterone replacement was initiated empirically. Within those days there was a partial return of ejaculatory pressure and a moderate shortening of the time span for the delayed erective reaction. The prostatic pain did not recur.

Husband and wife B Steroid Replacement

Once Mrs. B could accept the explanation for the onset of her husband’s sexual dysfunction, she was pleased to return to the role of an active sexual partner. The coital connection has continued regularly for the past three years with both members of the marital unit supported by steroid-replacement techniques.

In brief, for the sexually dysfunctional aging male, the primary concern is one of education so that both the man and his wife can understand the natural involutional changes that can develop within their established pattern of sexual performance.

Sex steroid replacement should be employed only if definite physical evidence of the male climacteric exists. As the newer techniques for establishing testosterone levels in blood serum become more widely disseminated, it will be infinitely easier to define and describe the male climacteric and therefore to offer testosterone replacement to those who need it, on a more definitive basis than an empirical diagnosis.

With effective dissemination of information by proper authority, the aging man can be expected to continue in a sexually effective manner into his ninth decade. Fears of performance are engendered by a lack of knowledge of the natural involutional changes in male sexual responsivity that accompany the aging process.

Really, the only factor that the aging male must understand is that loss of erective prowess is not a natural component of aging.

Statistical evaluation of the aging population and a consideration of treatment failure rates will constitute the following section. This material arbitrarily has been placed in the male rather than the female.

First, it was felt important to keep the statistical consideration of the aging marital units together, and second, because there were 56 males So years old or over in marital units accepted for treatment and only 37 wives 50 or older, it seemed appropriate to include the brief statistical discussion in the chapter reflecting the larger segment of the aging population.

Categories
Men's Health

The 11 Most Common Sexually Transmitted Diseases

The most important fact of sexually transmitted diseases is they are not contracted by people who have only one partner.

At the Clinic

Some clinics which specialize in the diagnosis and treatment of sexual diseases are free. Others charge for their services. Some are walk-in; others require an appointment to be made first. Many women opt to visit a health clinic rather than a family physician. They prefer the anonymity of their surroundings. When the infection is cured, there is no record in the family files.

Some STDs are “notifiable.” By law, they must be reported to the local health authorities. This varies from area to area, and from time to time. Other STDs are anonymous; a number instead of a name is used. Still, other diseases are confidential; name, address, and telephone number are kept in secret files. Again, this varies with the area and the time.

Some STDs have more than one name. Others have their names changed as more is discovered about them. They then get placed in their own special category; they no longer belong to the group they were originally designated. In much the same way, therapies and medications vary from clinic to clinic and from time to time. Though this can seem confusing, it shows an increase in medical knowledge of the disease. Also, environmental conditions and the endemic nature of the infection in one particular area are taken into account.

In towns and cities, there are hotlines to call for advice, help, and information. There are telephone tapes which are useful too. In isolated areas, look for notices in public locales, such as town halls, libraries, and restrooms. Consult the phone book. Entries might be under V for venereal disease or $ for STD. Above all, avoid delay in seeking help.

High Risk Behaviors

High risk sexual behavior includes:

  • Sex which is paid for.
  • Constant change of heterosexual partners.
  • Heterosexual anal sex which is unprotected.
  • Sex with an intravenous drug user.
  • “Tough” sex which causes lesions, bruises, bleeding.
  • Male sex (anal homosexual intercourse).

AIDS is transmitted by the HIV virus in blood, semen, and vagina fluids. It can be passed in skin sores and genital lesions too tiny to be seen with the unaided eye. It is also passed from mother to child in breast milk. Infected blood and semen contain the highest concentration of the virus. Vagina fluids have a lesser concentration. HIV may be present in sweat, saliva, and tears, but the concentrations are usually too weak for there to be any risk.

STDs, however, pass in very low concentrations. One germ can be enough. Studies suggest that syphilis and herpes are significant risk factors in the transmission of HIV. The sores of either disease can be on the mouth or inside the rectum, as well as on the genitals. In women, HIV is linked with a history of genital warts. It seems likely that STDs, which disrupt epithelial (lining) tissue, are important factors in the transmission of HIV. An appropriate way to avoid infection is to avoid direct contact with a partner’s semen, blood, or sores anywhere on the skin. Condoms provide some protection.

Gonorrhea

Gonorrhea is a bacterial infection which affects one million people each year in the United States. It is believed a further one million cases each year go unreported, because the disease is asymptomatic in 10 to 15 percent of men, and in 50 to 80 percent of women. Of the women with mild symptoms, 40 to 60 percent ignore them, believing that they are due to some other minor problem. The cervix is the most common site of gonorrhea.

Symptoms appear 3 days to 2 weeks after sexual contact. There is a thick, yellowish discharge. The cervix looks red, with small bump-like pits which are erosions. The urine tract often becomes infected, with the classic symptoms of UTI: stinging pain, frequency, and urgency. The infection can spread, to Skene’s and Bartholin’s glands. With oral sex, gonorrhea can spread from the penis to the throat, with sore throat and swollen glands, or it is asymptomatic. Discharge from an infected vagina or anal sex can infect the rectum with itching anus and discharge.

Untreated gonorrhea can lead to pelvic inflammatory disease (PID). Some 1 to 3 percent of women develop “disseminated gonorrhea,’ which spreads throughout the system. It can cause arthritis and, in rare cases, heart disease. The infection can be passed to a baby during birth, causing serious infection and possible blindness. Therapy is by antibiotics. Protect the cervix.

Syphilis

The corkscrew shaped bacteria of syphilis penetrate the skin of the vulva and within 30 minutes reach the glands in the groin. Thirty-six hours after infection, the bacteria have doubled in number. They double again every 30 hours. It takes an average 3 weeks (10 to 50 days) for the first symptoms to appear. By then, there are countless bacteria in the blood stream.

The first symptom is a chancre, an ulcer which starts as a pimple and then develops into an open sore with a hard rim. It is painless and self-healing. Once the sore disappears, bacteria travel in the blood, rapidly multiplying. Second stage syphilis occurs 2 to 6 weeks later. The symptoms include a skin rash over the body, swollen glands, and a flu-like condition; but often the disease is asymptomatic. Syphilis continues to wreak its havoc in the vital organs. In later years, the tertiary (third) stage is devastating: heart and brain disorders, joint inflammation, and sometimes early death.

Only about 10 percent of women who get chancres notice them. They can be hidden in the folds of the labia, under the hood of the clitoris, inside the vagina or rectum, on the cervix itself. The bacteria enter through any tiny skin lesion. The sores can appear anywhere, the most usual places being the mouth, nostril, tongue, even the finger. Avoid sexual contact if sores appear on any skin parts. The same applies to a partner.

Antibiotics destroy the bacteria of syphilis. Regular blood tests are necessary for the next two years to check for lingering germs. Keep all follow-up appointments to ensure that the disease has finally gone. Syphilis is 3 times more common in men than women; it is rare in female homosexuals. It can be passed to the fetus after the 20th week of pregnancy, so a blood test for syphilis is now a routine part of prenatal care.

Anal Sex

Anal sex carries specific health risks for all lovers, be they heterosexual or homosexual. Faeces contain highly infectious matter. The walls of the rectum are only a few cells thick. They are not designed to resist the pressure of a thrusting penis. They tear easily, and microscopic bleeding occurs. If the penis is not washed immediately after anal sex, whatever germs are in the bowel are thrust directly into the vagina. Infected semen, blood, or faeces can then pass directly into the blood system. Repeated attacks of yeast overgrowth can also occur this way.

Whatever the moral stance, hygiene is top priority. The penis should not touch the vulva, nor should it ever enter the vagina straight from the bowel. Hands, particularly fingernails, are an added danger in anal sex. Wiping with a tissue is not enough. Penis, hands, mechanical toys, all must be thoroughly scrubbed. It is strongly recommended a condom be used during anal sex, and immediately discarded afterwards.

Oral Sex

Specific micro-organisms inhabit the mouth, just as they inhabit other body orifices (openings). They rarely cause problems within their natural ecology. If they are transmitted to other orifices, they can cause infection. One typical example is a harmless bacteria of the mouth which can come in contact with the penis. The germs enter the urinary tract, and cause male UTI.

The membranes which line the mouth are naturally subjected to tiny lesions. It has been estimated that there is gum bleeding after brushing the teeth in at least one-third of any given population. Small ulcers can be present at the sides of the mouth. The tongue can be sore for a variety of reasons. All these factors can make the mouth an “unsafe” place for sex.

Diseases known to be transmitted by oro-genital infection are: the herpes virus cold sore, yeast infections, AIDS, gonorrhoea of the throat, and syphilis chancre of the lips. At least two cases of AIDS have been contracted this way. It would seem unlikely that a woman would wish to kiss a partner with a sore on the mouth, or that she would perform oral sex on a penis with a “drip”. Yet all infections have an incubation period. There is a time lapse between contracting a disease and the appearance of symptoms. Incubation periods vary widely with different STDs; they can take years for AIDS. With a new partner, the incubation period must be taken into account.

In some cases, both partners are asymptomatic. There are no signs of disease to remind lovers that oral sex can be hazardous. Avoid direct mouth contact with semen. Where there is high risk sexual activity, one option is to completely avoid oro-genital sex. If this is unacceptable, wait until a new partner has been tested and is known to be infection-free.

Tricky Trichomonas

Trichomonas vaginalis, or trich, is caused by a one-celled protozoan which grows rapidly within the vagina. Some women have an immediate and painful reaction to trich. Many more have asymptomatic trich; it is often only found if there are tests for other problems. The symptoms include a thin, foamy discharge which is yellow, green, or grey; there is intense itching and soreness, especially if the vulva is scratched. Trich can infect the urinary tract, causing burning, urgency and frequency. No tiny, one-celled creature should be able to cause such misery. But it does.

Trich can be passed on damp material: towels, bathing suits, washcloths, and toilet seats. This is rare. In most cases, it is transmitted by direct sexual contact. Metronidazole in Flagyl destroys trich. It has side effects, and should not be taken if there is any risk of pregnancy. A partner must be treated. Eschew douches and tampons. Avoid a flare-up recurrence by following the same “cool and dry” regime as for yeast overgrowth.

Vaginitis

There are many other organisms that can attack the area. They come under the generic terms nonspecific vaginitis and vulvitis. Nonspecific refers to conditions in which the cause is uncertain. They may be due to sexual infection, or they may not. The symptoms are often the same as for yeast and trich, with a profuse, foul-smelling discharge, intense itching, soreness, and in some cases, severe pain. Again, like yeast and trich, none of these attacks seem to affect the cervix. Yet they can cause real misery, and greatly reduce the quality of life.

Have a test for diabetes or a prediabetes condition first. Check diet and general health; try to boost the immune system by getting more rest, more profound sleep. Many women are run down and exhausted without realizing how deeply tired they are. Once yeast and trich are ruled out, a course of antibiotics may be the answer, though yeast overgrowth may then have to be treated. If attacks of vaginitis or vulvitis do recur, be extra scrupulous with genital hygiene. Keep the entire area cool and dry.

Chlamydia

Chlamydia is the most common STD in the U.S. today, with as many as 4 million new cases each year. It causes about half the known cases of NGU (non-gonococcal urethritis) in men. It breeds on the cervix in women. The symptoms are often mild and frequently go unnoticed. They are the same symptoms as for gonorrhea and can be confused with it. However, they appear a little later, within 1 to 3 weeks of sexual contact. More rarely, Chlamydia can be passed by a hand infected with the discharge from parent to baby.

If left untreated, chlamydia can lead to PID and infertility. Tests involve taking swabs from the cervix and culturing a specimen. The antibiotic of choice is tetracycline. Protect the cervix.

Herpes

The first attack of the herpes virus is the most painful and takes the longest time to heal. Within 2 to 20 days after infection, there is a mild tingling or itching. This can be on the labia, clitoris, or vagina opening; more rarely on the vagina wall, the cervix, the buttocks, thighs, or anus. It develops into one or more watery, painful blisters in the next few days. There can be burning or pain on urination, with swollen lymph nodes in the groin. There is an increase in discharge, or a feeling of pressure in the pelvic area. In some cases, the entire body reacts with flu-like symptoms: fever, headache, and chills.

Ninety percent of women develop sores on the vagina and cervix during a first infection. The blisters burst quickly, and shed highly contagious viruses everywhere. The now-empty blisters turn into shallow ulcers, which can be painful. The ulcers form into crusts, which heal spontaneously within 1 to 5 weeks. Visit the physician as soon as the symptoms appear. At an early stage, diagnosis can be made by sight alone. Help can begin immediately, but a culture test is very expensive.

At least 5 types of herpes virus are known to affect humans. The Epstein Barr virus and-cytomegalovirus causes infectious mononucleosis, also known as glandular fever. The varicella virus causes chicken pox in children, and shingles in adults. There are 2 types of herpes simplex virus. HSV 1 causes cold sores on the lips or nose, also called fever blisters. HSV 2 causes genital ulcers, also called genital herpes.

By adulthood, most people have been infected with the cold sore virus, HSV 1. They develop antibodies against it, and only a few actually get cold sores. Fewer adults have HSV 2 antibodies because the virus is spread by sexual contact. The findings of a recent study suggest that 99 percent of prostitutes have HSV 2 antibodies in their blood, compared with 3 percent of nuns and 29 percent of women in a committed relationship.

About 50 percent of those with HSV 2 have no symptoms. The recent increase in genital herpes is thought to be partly due to this, and partly due to an increase in the practice of oro-genital sex. In some cases, both HSV 1 and HSV 2 cause genital herpes. If suffering from a cold sore, avoid kissing, and any facial or genital contact. This applies to a partner as well.

Not all HSV 2 die after a first attack. The virus coats itself in the person’s own protein substance and retreats along nerve endings to the base of the spine. Here it sets up a permanent home, staying inactive for varying lengths of time. When the virus becomes active again, it usually returns to the same place as the previous attack. Recurring outbreaks can be virulent and painful, or very mild. If mild, a woman may be unaware that she is shedding highly contagious germs.

HSV 2 is particularly dangerous for women. It is linked with cancer of the cervix, The virus can cause miscarriage in the first 3 months of pregnancy. If shed during birth, 1 in 2 babies will be infected. Two out of 3 of those infected babies will die. Half the others suffer brain damage, or visual defects. These horrors are now avoided by Caesarian birth. The baby is lifted from the uterus and thus avoids contact with the virus.

As yet, there is no drug to destroy the herpes virus. The drug acyclovir helps reduce the pain of an attack; it may even lessen the number of recurrences. One of the miserable factors of herpes is the permanent risk of passing on the disease. Some physicians believe that this is only during the active phase; others strongly disagree. An infected person cannot be free of this worry.

Genital Warts

Molluscum Contagiosum: There are two kinds of warts, simple and genital. Both can infect the genitals; it is crucial to recognize the difference. Simple warts are the kind which appears on the hands of children. They are small, dimpled papules, which look like spots with a drop of pearly fluid inside. They are highly contagious, as their Latin name shows. They can be transmitted to the genitals by self or partner from warts on the hands and elsewhere. The virus enters the skin through invisible lesions which occur during sexual activity. The warts appear some 30 days after contact. Attacks of simple warts on the genitals are rare, being most likely in the teens and 20 to 30 age group.

If the penis is infected with simple warts, some men try self therapy. This is not advisable for women. Simple warts can be painful if rubbed, otherwise a woman is unaware of them. They are not life-threatening, nor do untold damage, but they are highly contagious. Visit the physician or clinic. Therapy varies.

Human Papilloma Virus: HPV is specific to the genital area. It is transmitted by direct sexual contact. The warts appear 3 weeks to 3 months after contact, but the incubation time can be up to 8 months, even more. The warts can be single; usually, they grow in clusters like grapes. With their raised, bumpy tops, they look like miniature cauliflowers. They grow on the labia lips or anus, inside the vagina, or on the cervix. In many cases, they are asymptomatic, and the woman is unaware that she is infected.

The warts are painless, but easily irritated by rubbing, and sometimes they itch. If there has been anal contact, they can grow inside the rectum and around the anus. More rarely with oral contact, they infect the linings of the mouth. If the warts breed in colonies on the cervix, the disease may not be detected until a Pap smear is done. Women with HPV have a five times higher risk of cancer of the cervix.

Larger warts, especially on the cervix, maybe vaporized by laser therapy, but it is difficult to know if they have all been destroyed. The healing process takes 6 weeks. Repeat therapy is necessary if they flare up again; avoid losing patience as laser therapy usually works. Other therapies include burning the warts off by electric cautery, or freezing them with dry ice. The physician then snips them off. External warts can be painted with the drug podophyllin. It takes 3 or 4 weekly treatments for the warts to dry up and drop off.

HPV infection is also called condyloma. The prescription drug Condylox has just been made available for home treatment, which means that patients no longer need to have a physician apply the therapy. At least 56 different types of the virus have been identified.

Hepatitis

Hepatitis A and B are caused by virus infection of the liver. The virus breeds in waste matter from the bowel and is common where there is poor sanitation. It is passed in contaminated food and drink; less usually, by sexual contact; more rarely, by transfusions of infected blood. Hepatitis is on the increase, probably due to more foreign travel. When visiting areas with poor sanitation, observe strict personal hygiene. Drink bottled water, eschew ice cubes. Avoid anal and oral sexual contact.

The symptoms of both A and B are the same: fever, nausea, headache, fatigue, loss of appetite, and chills. Jaundice shows as a yellow tinge to the skin, fingernails, and whites of the eyes about a week later. Urine can be dark in colour; stools almost whitish. A few people are asymptomatic. With hepatitis A, the symptoms are mild. The defence system builds immunity to the virus, but it remains in the blood and can be transmitted.

The hepatitis B virus (HBV) produces severe symptoms, which start suddenly 1 to 6 months after contact. If liver damage is extensive, death occurs in 5 to 20 percent of cases. The B virus is transmitted in blood and blood products during sexual contact: semen, vagina secretions, saliva, and faeces are suspect. It is also passed by IV drug users sharing infected needles. The incidence of HBV is rising rapidly, perhaps due to more foreign travel and IV drug use. Male homosexuals, heterosexuals with multiple partners, travellers, and drug addicts are high risk groups.

AIDS

AIDS stands for Acquired Immune Deficiency Syndrome.

Acquired: it is passed on, but not inherited.
Immunodeficiency: the immune system grows weak and deficient.
Syndrome: a group of symptoms of which the cause is unknown.

However, it is now known that AIDS is caused by the human immunodeficiency virus (HIV). The word AIDS is still used to avoid confusion. The virus does not kill, but it damages the immune system, leaving the person vulnerable to rare infections and cancers which are life-threatening. If death occurs, it is not from AIDS, but from one of these opportunistic diseases.

HIV is transmitted in body fluids: blood, blood products, semen, vagina secretions, and breast milk. It does not appear to be easily transmitted in saliva.

The Future

Scientists using an experimental AIDS vaccine have succeeded in changing the way the body fights the AIDS virus. The discovery could open the door to new ways of treating the disease. By giving the vaccine to 30 men and women infected with HIV, researchers found that they were able to prompt the immune systems of most in the group into mounting a more sophisticated counterattack against the virus. It is too early to know if this response will help HIV-infected people to survive the ravages of the disease.

The study’s results counter the long-standing and pessimistic conviction of many AIDS researchers that there is little to be done to improve upon the immune system’s battle against the HIV virus.

New therapies such as the use of the antiviral drug AZT early in infection and inhaled pentamidine to prevent AIDS-caused pneumonia will delay the time when HIV infection develops into full-blown AIDS.

Categories
Men's Health

Sexual Function

To establish at least a minimum of patient screening, at the onset of the clinical treatment program, no units were accepted in therapy unless the complaining partner in the couple (e.g., the impotent male or the non-orgasmic female) had a history of at least six months of prior psychotherapeutic failure to remove the symptoms of sexual dysfunction. Very soon this proved to be a poorly contrived standard, of little screening value.

As should have been apparent at the onset, there was no secure way of establishing the functional effectiveness of the prior therapeutic program. How determined and well oriented was the therapist, how cooperative or fully responsible was the patient? After two years this original standard was abandoned in favor of that currently in effect.

Sexual Screening

A reasonably effective method of screening has been substituted by requiring that no patients be accepted at the Foundation unless they have been referred by the authority. As the authority, the Foundation accepts physicians, psychologists, social workers, and theologians.

Beyond screening the patients for appropriate referral to the Foundation, the referral source further is asked to provide available details of psychosocial background relevant to the husband and wife’s sexual dysfunction.

A telephoned report is made to the referring authority describing husband and wife’s progress (or lack of it) during or immediately following the acute phase of treatment at the Foundation. The well-informed authority then can provide the most important reinforcement for newly acquired patterns of sexual interaction for the couple once removed from the Foundation’s direct control by the termination of the acute phase of therapy.

In many instances, patients in established psychotherapeutic programs have been referred for removal of symptoms reflecting a somewhat broad area of distress in which sexual inadequacy is only a part. After their two weeks at the Foundation, these couples are, of course, returned to referring authority to continue their established treatment programs.

Obviously, the referring authority, before continuing in therapy with his patient, is briefed in detail as to the couple’s response to its Foundation exposure. The screening process as currently constituted has several aims, all obviously selective in nature.

Symptoms of Sexual Inadequacy

Primarily, control which prevents referral of major psychopathology is presumed. In other words, the psychoneurotic is acceptable, but not psychotic.

It should be emphasized that the reversal of symptoms of sexual inadequacy in psychoneurotic patients is indeed a significant portion of the Foundation’s objectives. Acceptance of this role by the Foundation is based on the premise that the reversal of particularly troublesome sexual symptoms may speed the progress of a psychoneurotic patient within the greater context of his established and broader-based psychotherapy.

However, the majority of the couples contending with sexual dysfunction do not evidence psychiatric problems other than the specific symptoms of sexual dysfunction. Socio-cultural deprivation and ignorance of sexual physiology, rather than psychiatric or medical illness, constitute the etiologic background for most sexual dysfunction.

Therefore, when a couple is properly educated in sexual matters, and their specific symptoms are reversed, there is no need for further psychotherapy unless the extensive duration of the distress has created psychosocial complications no longer directly related to the sexual dysfunction.

Other areas of selective screening for information vital to the therapeutic program center on such questions as:

  1. Are both members really interested in reversing their basic dysfunctional status? If one member of the unit simply has no interest whatsoever in reversing the symptomatology of sexual dysfunction in the marital relationship, the unit probably needs legal rather than medical or behavioral advice. The chances of reversing sexual dysfunction under the circumstances of total disaffection for a marital partner are negligible.
  2. What, if anything, is known of the couple’s adjustment or maladjustment to its social community?
  3. Do the referred members of the couple understand the programs, procedures, and policies of the Foundation? If not, it is suggested that the local authority, quietly briefed in advance by the Foundation’s professional staff, present the information in more specific detail to his patients.
  4. What is the couple’s basic financial picture? Should the Foundation offer the patients an adjusted fee scale or free care?

Sexual Therapy Commitment

The original research premise emphasized the fact that the positive reversal of symptoms of sexual inadequacy during the acute phase of the treatment program was not of great importance. If there were to be any clinical claim for positive effect in the Foundation’s concentrated approach to symptom reversal, the clinical results would have to be judged in retrospect over a significant period of time, not at the termination of the acute phase of therapy.

Therefore, the policy of five years of follow-up for couples after the termination of the rapid-treatment phase of the program became an integral part of research standards. Failures to reverse symptoms are, of course, considered most significant.

Little clinical value can be established for any therapeutic program, regardless of the length of its ongoing treatment phase, if the results are not evaluated in long-term follow-up after the termination of the acute phase of therapy. The abiding guide to treatment value must not be how well the patients do under authoritative control but how well they do when returned to their own cognizance without therapeutic control.

This result finally must place the mark of clinical failure or success upon the total therapeutic venture.

Individual members of couples seen in treatment must agree to cooperate with five years of follow-up after the termination of the acute phase of the therapy program. They fully understand.

The Foundation’s basic premise that success in a reversal of the symptoms of sexual dysfunction means little during the two weeks of intensive treatment unless the symptom reversal is maintained for at least the first five years after separation from direct Foundation influence.

Success in the maintenance of symptom reversal for this length of time does provide some sense of permanency in the continuing effectiveness of the couple’s sexual functioning.

Those couples whose acute treatment phase was judged inadequate or a failure arbitrarily have not been placed in the five-year follow-up program. This type of follow-up would indeed have been a study of major importance, but such continuing interrogation certainly could have interfered seriously with other clinical approaches designed to relieve the unit’s problems of sexual dysfunction.

The therapy concepts and clinical procedures depict the basic methodology of co-therapist interaction, first, between team members, and second, directed toward husband and wife of the sexually dysfunctional marital, unit. Jules Masserman has so aptly described psychotherapy as “anything that works.” This “works” in a healthy percentage of cases.

Categories
Men's Health

Male Impotence Causes

A typical history of an acute episode of alcohol consumption as an etiological factor in the onset of secondary impotence is classic in its structural content. The clinical picture is one of acute psychic trauma on a circumstantial basis, rather than the chronic psychosocial strain of years of steady attrition to the male ego as described in the case history for the premature ejaculator.

There has been a specific history of onset of symptoms of secondary impotence as a direct result of episodes of acute alcoholic intake in 35 men from a total of 213 men referred with a complaint of secondary impotence.

The onset of secondary impotence in an acute alcoholic episode is so well known that it almost beggars description. A composite example is that of a relatively “successful” male aged 35-55, college graduate, working in an area which gears productive demand more to mental than physical effort.

The perfect environmental situation for onset of secondary impotence is any occupational hazard where demands for high levels of psychosocial performance are irrevocably a part of the nine-to-five day and frequently carry over into an evening of professional socializing.

Alcohol Impotence

Mr. A is a man with a habit of alcohol before dinner, frequently a few glasses of wine with his meals, and possibly a whisky. Alcohol intake at lunch is an integral part of his business as well.

In short, consumption of alcohol has become a part of his life.

This man and his wife leave home one night for a party and alcohol is available in large quantity. Somewhere in the late evening, the party comes to an end. Mr. A has had entirely becomes tipsy and so his wife drives them home for safety’s consideration.

His wife retires to the bedroom, and with a sense of vague irritation, a combination of a sense of personal rejection and a residual of her social embarrassment, prepares for bed. Mr. A has stumble but with the aid of a strong banister and even stronger nightcap, manages to arrive at the bedroom door. Suddenly he felt that his wife is indeed fortunate tonight, for he is prepared sexually satisfied her.

Alcohol Hangover

It never occurs to him that all she wants to do is go to bed and avoid a quarrel at all costs. He jumped into the bed, moves to meet his imagined commitment, and nothing happens. He has simply had too much alcohol.

Dismayed and confused both by the fact that no erection develops and that his wife obviously has little or no interest in his gratuitous sexual contribution, he pauses to resolve this complex problem and immediately falls into deep, anesthetized slumber.

Next day, he is further traumatized by the symptoms of an acute hangover. He surfaces later in the day with the concept that things are not as they should be. The climate seems rather cool around the house. He can remember little of the prior evening’s festivities except his deeply imbedded conviction that things did not go well in the bedroom. He is not sure that all was bad but he also is quite convinced that all was not good.

Obviously he cannot discuss his problem with his wife, she probably would not speak to him at this time. So he putters and mutters throughout the evening and goes to bed early to escape. He sleeps restlessly only to face the new day with a vague sense of alarm, a passing sense of frustration, and a sure sense that all is not well in the household this Monday morning.

He pondered about it over a drink or two at lunch and another, and while contending with traffic on the way home from work, decides to check out this evening the little matter of sexual dysfunction, which he may or may not have imagined.

Sexual dysfunction within 48 hours!

If the history of this reaction sequence is taken accurately, it will be established that Mr. A does not check out the problem of sexual dysfunction within 48 hours of onset, as he had decided to do on his way home from work. He arrives home, finds the atmosphere still markedly frigid, makes more than his usual show of affection to the children, retires to the security of the cocktail hour, and goes to supper and to bed totally lacking in any communicative approach to his frustrated, irritated marital partner.

Tuesday morning, while brushing his teeth, Mr. A has a flash of concern about what may have gone wrong with his sexual functioning after the party night. He decides unequivocally to check the situation out tonight.

Instead of thinking of the problem occasionally, his concern for “checking this out” becomes of paramount importance. On the way to work and during the day, he does not think about what really did go wrong sexually because he does not know. Rather he worries constantly about what could have gone wrong.

Needless to say, there is resurgence of concern for sexual performance during the afternoon hours, regardless of how busy his schedule is.

Mr. A leaves the office in relatively good spirits, but thoroughly aware that “tonight’s the night.” He does have vague levels of concern, which suggest that a little relaxation is in order; so he stops at his favorite tavern for a couple of drinks and arrives home with a rosy glow to find not only a forgiving, but an anticipatory, wife, ready for the reestablishment of both verbal and sexual communication that a drink ‘or two together before dinner can bring.

Probably for the first time in his life, he approaches his bedroom on Tuesday night in a self-conscious “I’ll show her” attitude. Again there has been a little too much to drink-not as much as on Saturday night, but still a little too much.

And, of course, he does show her. He is so consumed with his conscious concern for effective sexual function (the onset of his fears of performance) that, aided by the depressant effect of a modest level of alcoholic intake (modest by his standards), he simply cannot “get the job done.”

When there is little or no immediate erective reaction during the usual sexual preliminaries, he tries desperately to force the situation-in turn, anticipating an erection, then wildly conscious of its abscence, and finally demanding that it occur. He is consciously trying to will sexual success, while subjectively watching for tumescence. So, of course, no erection.

While in an immediate state of panic, as lie sweats and strains for the weaponry of male sexual functioning, he simultaneously must contend with the added distraction of a frightened wife trying to console him in his failure and to assure him that the next night will be better for both of them.

Sexual Incompetence

Both approaches are equally traumatic from his point of view. He hates both her sympathy and blind support which only serve to underscore his “failure,” and reads into his wife’s assurances that probably he can do better “tomorrow” a suggestion that no longer can he be counted on to get the job done sexually when it matters “today.”

A horrible thought occurs to Mr. A. He may be developing some form of sexual incompetence. He has been faced with two examples of sexual dysfunction. He is not sure what happened the first time, but he is only too aware this night that nothing has happened. He has failed, miserably and completely, to conduct himself as a man.

He cannot attain or maintain an erection.

Further, Mr. A knows that his wife is equally distressed because she is frantically striving to gloss over this marital catastrophe. She has immediately cast herself in the role of the soothing, considerate partner who says, “Don’t worry dear, it could happen to anyone,” or “You’ve never done this before, so don’t worry about it, dear.”

In the small hours of the morning, physically exhausted and emotionally spent from contending with the emotional bath her husband’s sexual failure has occasioned, she changes her tune to “You’ve certainly been working too hard, you need a vacation,” or “How long has it been since you have had a physical checkup?” (Any of a hundred similar wifely remarks supposed to soothe, maintain, or support are interpreted by the panicked man as tacit admission of the tragedy they must face together: the progressive loss of his sexual functioning.)

From the moment of second erective failure,

72 hours after the first erection failure, this man may be impotent.

In no sense does this mean that in the future he will never achieve an erection quality sufficient for intromission.

Occasionally he may do so and most men do. It does mean, however, that any suggestion of wifely sexual demand either immediate in its specific physical intensity or pointing coyly to future sexual expectations may produce pressures of performances quite sufficient to reduce Mr. A to and maintain him in a totally no erective state.

In brief, fears of sexual performance have assumed full control of his psychosocial system.

Mr. A thinks about the situation constantly. He occasionally asks friends of similar age group how things are going, because, of course, any male so beleaguered with fears of sexual failure is infinitely desirous of blaming his lack of effective function on anything other than himself, and the aging process is a constantly available cultural scapegoat.

Sexual Approach

He finds himself in the position of the woman with a lifetime history of non orgasmic return who contends openly with concerns for the effectiveness of her own sexual performance and secretly faces the fear that in truth she is not a woman. In proper sequence he does as she has done so many times.

He develops ways and means to avoid sexual encounter.

He sits fascinated by a third-rate movie on television in order to avoid going to bed at the usual time with a wife who might possibly be interested in sexual expression. He fends off her sexual approaches and jumps at anything that avoids confrontation as a drowning man would at a straw.

His wife immediately notices his disinclination to meet the frequency of their semi established routine of sexual exposure. In due course she begins to wonder whether he has lost interest in her, if there is anyone else, or whether there is truth in his most recent assertion that he couldn’t care less about sex.

For reassurance that she is still physically attractive, the concerned wife begins to push for more frequent sexual encounters, the one approach that the self-pressured male dreads above all else.

Obviously, neither marital partner ever communicates his or her fears of performance or the depth of their concerns for the sexual dysfunction that has become of paramount importance in their lives. The subject either is not discussed, or, if mentioned even obliquely, is hastily buried in an avalanche of words or chilled by painfully obvious avoidance.

Sexual Anxiety

Within the next 3 months, Mr. A has to fail at erective attainment only another time or two before both husband and wife begin to panic.

She decides independently to avoid any continuity of sexual functioning, eliminate any expression of her sexual needs, and be available only should he express demand, because she also has developed fears of performance.

Her fears are not for herself, but for the effectiveness of her husband’s sexual functioning.

She goes to great lengths to negate anything that might be considered sexually stimulating, such as too-long kisses, handholding, body contact, caressing in any way. In so doing she makes each sexual encounter much more of a pressured performance and therefore, much less of a continuation of living sexually, but the thought never occur to her. All communication ceases.

Each individual keeps his own counsel or goes his own way. The mutual sexual stimulation in the continuity of physical exposure, in the simple physical touching, holding, or even verbalizing of affection, is almost totally withdrawn.

The lack of communication that starts in the bedroom rapidly spreads through all facets of marital exchange: children, finances, social orientation, mothers-in-law, whatever.

In short :
Sexual dysfunction in the marital bed, created initially by an acute stage of alcoholic ingestion, supplemented at the next outing by ah “I’ll show her” attitude and possibly a little too much to drink can destroy the very foundation of a marriage of 10 to 30 years duration.

As the male panics, the wife only adds to his insecurity by her inappropriate verbalization, intended to support and comfort but interpreted by her emotionally unstable husband as immeasurably destructive in subjective content.

The dramatic onset of secondary impotence following an in stance of excessive alcohol intake is only another example of the human male’s extreme sensitivity to fears of sexual performance.

In this particular situation, of course, the onset of fears of performance was of brief but dynamic duration as opposed to those in the preceding example of the premature ejaculator whose fears of performance developed slowly, stimulated by continued exposure to his wife’s verbal denunciation of his sexual functioning.

Discussed above are examples of combinations of psychological and circumstantial factors that contribute the highest percentage of etiological input to the development of secondary impotence. Continuing through the listing of major influences there remain environmental, physiological, and iatrogenic factors.

In the final analysis:
Regardless of listing category, secondary impotence is triggered by combinations of these etiological factors rather than by any single category with the obvious exception of psychosocial influence. Once onset of erective failure has been recorded, regardless of trigger mechanism, involved, the individual male’s interpretation of or reaction to functional failure must be dealt with on a psychogenic basis.

The etiological factors recorded above are little more than categorical conveniences. From his initial heterosexual performance through the continuum of his sexual expression, every man constantly assumes a cultural challenge to his potency.

How he reacts to these challenges may be influenced directly by his psychosocial system, but of particular import is the individual susceptibility of the man involved to the specific pressures of the sexual challenge and to the influences of his background.

When considering etiological influences that may predispose toward impotence, it always should be borne in mind that most men exposed to parallel psychosexual pressures and similar environmental damage shrug off these handicaps and live as sexually functional males.

It is the factor of susceptibility to negative psychosocial input that determines the onset of impotence. These concepts apply to primarily as well as secondarily impotent men.

When considering environmental background as an etiological factor in secondary impotence, the home, the church, and the formative years are at center focus.

What factors in or out of the home during the formative years tend to initiate insecurity in male sexual functioning?

The preeminent factor in environmental background reflecting sexual insecurity is a dominant imbalance in parental relationships dominant, that is, as opposed to happen stance, farcical, or even fantasized battles for family control.

Secondary, but still of major import is the factor of homosexuality, which is to be considered in the environmental category. In no sense does this placement connote professional opinion that homophile orientation is considered purely environmental in origin.

Since homosexual activity may have derogatory influence upon the effectiveness of heterosexual functioning, the subject must be presented in the etiological discussion. The disassociations developing from homophile orientation are considered in the environmental category only for listing convenience.

Categories
Women's Health

Impotent and Female Orgasm

Although emphasis has been placed upon the role of premature ejaculation in the etiology of primary orgasmic dysfunction, primary or secondary impotence also contributes. Again the basic theme of man and woman coital interaction must be emphasized.

If there is not a sexually effective male partner, the female partner has the dual handicap of fear for her husband’s sexual performance as well as for her own.

If there is no penile erection there will be no effective coital connection.

Frequently women married to impotent men cannot accept the idea of developing a masturbatory facility or being manipulated to orgasm as a substitute for tension release.

However, if there has been a masturbatory pattern established before coital inadequacy assumes dominance, most women can return to this sexual outlet. In this situation, there is sufficient dominance of the previously conditioned biophysical structure to overcome negative input from a psychosocial system distressed by sexual performance fears.

But if there have been no previous substitute measures established, many women cannot turn to this mode of relief once impotence halts effective coital connection. In this situation, the psychosocial structure, unopposed by prior biophysical conditioning, assumes the dominant influence in the woman’s sexual response pattern.

Mr. and Mrs. D

were referred to the treatment of orgasmic inadequacy after four years of marriage. When seen in therapy she was 27 and her husband 43 years old. He had been married twice previously. There were children of both marriages and none in the current marriage.

The husband also was sexually dysfunctional in that he was secondarily impotent.

His second marriage had been terminated due to his inability to continue the effective coital connection. Although, when the unit was seen in consultation the marriage had been consummated, coitus occurred only once or twice a year.

Mrs. D’s background reflected somewhat limited financial means. Her father had died when the three siblings were young, and the family had been raised by their mother, who worked while the grandmother took care of the children.

Clothes were hand-me-downs, food the bare essentials. Her education had of necessity terminated with high school, and she worked as a receptionist in several different offices befo3e her marriage. She continued to live at home while working and contributed to what salary she made to help with the family’s limited income.

Mrs. D met her future husband when he visited the office where she worked. He invited the young woman to lunch. She accepted and married him four months later without knowledge of his sexual inadequacy, although she had been somewhat puzzled by his lack of forceful sexual approach during the brief courtship.

Mrs. D’s own sexual history had been one of a few unsuccessful attempts at masturbation, numerous petting episodes with boys in and out of high school, but no attempted coital connection. She had never been orgasmic.

Her husband had inherited a large estate and his financial situation certainly was the determining factor in his wife’s marital commitment. Since the girl had been distressed by a family background of genteel poverty, she felt the offer of marriage to be her real opportunity both to escape her environment and to help her two younger siblings.

The wedding trip was an unfortunate experience for Mrs. D when she realized for the first time that her husband had major functional difficulties. She knew little of male sexual response beyond the petting experiences but did try to help him achieve an erection by a multiplicity of stimulative approaches in his direction. There was no success in erective attainment.

The marriage was not consummated until six months after the ceremony. In the middle of the night, Mr. D awoke with an erection, moved to his wife, and inserted the penis. She experienced mild pain but reacted with pleasure, feeling that progress had been made.

However, following the usual pattern of .a secondarily impotent male, progress was fleeting. As stated, there were only a few other coital episodes in the course of the four-year marriage. In these instances, she always was awakened from sleep by her husband when he awoke to find himself with an erection.

Quick Ejaculation

Then there was rapid intromission and quick ejaculation. There was no history of a successful sexual approach by her husband under her conscious direction, insistence, or stimulation. Mr. D tried repetitively during long-continued manipulative and oral-genital sessions to bring his wife to orgasm, without result.

When they were referred for treatment, neither husband nor wife described sexual activity outside the marriage.

There have been 193 women treated for primary orgasmic dysfunction during the past 11years. Basically, in this method of therapy, the sexually dysfunctional woman is approached through her sexual value system.

If its requirements are non-serving limited, unrealistic, or inadequate to the marital relationship-by suggestion she is given an opportunity, with her husband’s help, to manipulate her biophysical and psychosocial structures of influence until an effective sexual value system is formed.

 
Categories
Women's Health

Sexual Lubrication

Sexual Lubrication

Probably the most frequent cause for dyspareunia originating with symptoms of burning, itching, or aching is a lack of adequate production of vaginal lubrication with sexual functioning.

During attempted penile intromission or with long-maintained coital connection, there must be adequate lubrication or there will be irritative distress for either or both coital partners.

Adequate production of vaginal lubrication is for women the physiological equivalent of erective attainment for men and her representation to her male partner of psychological readiness for vaginal penetration.

A consistent lack of functional levels of vaginal lubrication is as near as any woman can come to paralleling a man’s lack of effective erection.

Inadequate development of vaginal lubrication has many causes, but by far the most common is lack of interest in the particular opportunity or identification with the involved sexual partner. Women must experience positive input from their sexual value systems if they are to respond repetitively and effectively to their mate’s sexual approaches.

Women who cannot think or feel sexually, those reflecting fears of sexual performance, and even those acting out a spectator’s role in sexual functioning, either do not develop sufficient vaginal lubrication to support a painless coital episode or, if penetrated successfully, they cease production of the necessary lubricative material shortly after intravaginal penile thrusting is initiated.

Deep Penetration

Additionally, women fearful of pain with deep vaginal penetration, fearful of pregnancy with any coital connection, fearful of exposure to social compromise, and fearful of sexual inadequacy are frequently poor producers of vaginal lubrication. Any Component of fear present in sexual experiences reduces the receptivity to sensate input, thereby blunting biophysical responsivity.

If there is insufficient vaginal lubrication, there may or may not be acute pain with full penile intromission, depending primarily upon the parity of the woman, but they’re usually will be vaginal burning, irritation, or aching both during and after coital connection.

There are tens of thousands of women who become markedly apprehensive at the onset of any definitive sexual approach, simply because they know severe vaginal irritation will be experienced not only during but frequently for hours after any significantly maintained coital connection.

Not to be forgotten as a specific segment among the legions of women afflicted by inadequate production of vaginal lubrication are those in their postmenopausal years. If they are not supported by adequate sex-steroid-replacement techniques, the production of vaginal lubrication usually drops off markedly as the vaginal mucosa routinely turns atrophic.

There well may be aching and irritation in the vagina for a day or two after a coital connection for women contending with this evidence of sex-steroid starvation.

A major category of women tending to be poor lubrication producers during coital connection is that a significant segment of the female population with overt lesbian orientation. Many women psychosexually committed to a homophile orientation attempt regularity of coital connection for socioeconomic reasons. Frequently, they may not lubricate well during heterosexual activity, although there usually is ample lubrication when they are directly involved in the homosexual expression.

In most instances:

Inadequate production of vaginal lubrication can be reversed with a definite therapeutic approach. Certainly, women burdened by a multiplicity of sex-oriented fears can be provided psychotherapeutic relief of their phobias and subsequently reversed with relative ease from their particular pattern of sexual inadequacy.

Those women with chronic vaginal itching and irritation can be protected from continuing dyspareunia because both infectious and chemical vaginitis are reversible under proper clinical control. Senile vaginitis responds in short order to adequate sex-steroid-replacement techniques.

There are only two major categories of women for whom the co-therapists have little to offer to constitute effective production of vaginal lubrication: first, women mated to men for whom they have little or no personal identification, understanding, affection, or even sexual respect; and second, homosexually oriented women practicing coition for socioeconomic reasons with no interest in their male companions as sexual partners.

Undesired Sex

As opposed to the symptoms of aching, irritation, or burning in the vagina, complaints of severe pain developed during penile thrusting provide the most difficulty in delineating between subjective and objective etiology.

Although many women register this type of complaint when seeking to avoid undesired sexual approaches, there are some basic pathological conditions in the female pelvis that can and do engender severe pain in response to active coital connection.

One of the difficulties in delineating the severity of the complaint of dyspareunia is to identify pelvic pathology of a quality sufficient to support the female partner’s significant complaints of painful coition. The pelvic residual from severe infection or pelvic implants of endometriosis usually is easily identified by adequate pelvic and rectal examinations.

These clinical entities will be discussed briefly in context later in the topic. Current attention is drawn to probably the most frequently overlooked of the major physiological syndromes creating intense pelvic pain during coital connection.

Categories
Women's Health

Sex, Pelvic Syndromes

One of the most obscure of pelvic pathological syndromes, yet one of the most psychosexually crippling, is traumatic laceration of the ligaments supporting the uterus. This syndrome was described clinically. Five women have been referred to the Foundation for relief of subjective symptoms of dyspareunia after referral sources had assured the husbands that there was no plausible physical reason for the constant complaint of severe pain with deep penile thrust.

These 5 postpartum women had severe broad ligament lacerations and were relieved of their distress by definitive surgical approaches, not by psychotherapy. Therefore, they do not represent a component of the statistical analysis of treatment for sexual dysfunction.

Three women reflecting the onset of dyspareunia after criminal abortion techniques also have been seen in consultation and are not reflected in the statistics of the sexual inadequacy study. Three more women have been seen in gynecological consultation for acquired dyspareunia after gang-rape experiences.

They also have not been an integral part of the sexual-dysfunction study. These clinical problems will be mentioned in context. When first seen clinically, women with traumatic lacerations of the uterine supports, acquired with delivery or by specific criminal abortion techniques, present complaints that commonly accrue from pelvic vasocongestion, dyspareunia, dysmenorrhea, and a feeling of being excessively tired.

These complaints are secondary or acquired in nature. The traumatized women consistently can relate the onset of their acquired dyspareunia to one particular obstetrical experience even from among three or four such episodes.

The basic intercourse distress arises with deep penetration of the penis. Women describe the pain associated with intercourse to be as if their husbands had “hit something” with the penis during deep vaginal penetration.

These involved women may note other physical irritations frequently seen with chronic pelvic vasocongestion, a constantly nagging backache, throbbing or generalized pelvic aching, and occasionally, a sense that “everything is falling out.”

These symptoms are made worse in any situation requiring a woman to be on her feet for an exceptional length of time, as a full day spent doing heavy housecleaning or working as a saleswoman in a department store.

Most women lose interest in any regularity of sexual expression when distressed by acquired dyspareunia. Handicapped by constant anticipation of painful pelvic stimuli created by penile thrusting, they also may lose any previously established facility for orgasmic return.

The basic pathology of the syndrome of broad ligament laceration is confined to the soft tissues of the female pelvis. The striking features of the pelvic examination are the position of the uterus and almost always in severe third-degree retroversion and the particularly unique feeling that develops for the examiner with manipulation of the cervix.

This portion of the uterus feels just as if it were being rotated as a universal joint. It may be moved in any direction, up, down, laterally, or on an anterior-posterior plane with minimal, if any, correspondingly responsive movement of the corpus and body of the uterus.

Even the juncture of the cervix to the lower uterine segment is ill-defined. The feeling is one of an exaggerated Hegar’s sign of early pregnancy, in which the cervix appears to move in a manner completely independent of the attached corpus.

In addition to the “universal joint” feeling returned to the examiner when moving the cervix, a severe pain response usually is elicited by any type of cervical movement. However, pain is primarily occasioned by pushing the cervix in an upward plane.

In the more severe cases, either in advanced bilateral broad ligament laceration or in a presenting complaint of five years or more in duration even mild lateral motion of the cervix will occasion a painful response. During the examination, the retroverted uterus appears to be perhaps twice increased in size.

Pressing against the corpus in the cul-de-sac to reduce the third-degree retroversion also will produce a marked pain response. When the examiner applies upward pressure on the cervix or pressure in the cul-de-sac against the corpus, the patient frequently responds to the painful stimulus by stating, “It’s just like the pain I have with intercourse.”

A detailed obstetrical history is a salient feature in establishing the diagnosis of the broad ligament laceration syndrome. The untoward obstetrical event creating the lacerations may be classified as surgical obstetrics, as an obstetrical accident, or even as a poor obstetrical technique.

Occasionally, women with both the positive pelvic findings and the subjective symptoms of this syndrome cannot provide a positive history of obstetrical trauma, but this situation does not rule out the existence of the syndrome.

Many women are unaware of an unusual obstetrical event because they were under advanced degrees of sedation or even full anesthesia at the time.

Precipitate deliveries, difficult forceps deliveries, complicated breech deliveries, and postmature-infant deliveries are all suspect as obstetrical events that occasionally contribute to tears in the maternal soft parts.

If these tears are established in the supports of the uterus (broad ligaments) the immediate postpartum onset of severe dyspareunia can be explained anatomically and physiologically. It is important to emphasize clinically that although a woman may not be able to describe a specific obstetrical misfortune in her history, she well may be able to date the onset of her acquired dyspareunia to one particular obstetrical event.

Three cases have been seen in which a criminal abortion was performed by extensively packing the vaginal barrel, leading ultimately to dilation of the cervix and expulsion of uterine content.

These extensive vaginal-packing episodes created tears of the broad ligaments, completely parallel to those occasioned by actual obstetrical trauma. Each woman, although unaware of the etiological significance, dated the onset of the symptoms of acquired dyspareunia to the specific experience with the vaginal-packing type of abortive technique.

 
Categories
Overall Health

5 Tips to Protect Your Joints

What are the Joints?

joint or articulation is the connection made between bones in the body which link the skeletal system into a functional whole. They are constructed to allow for different degrees and types of movement. Some joints, such as the knee, elbow, and shoulder, are self-lubricating, almost frictionless, and are able to withstand compression and maintain heavy loads while still executing smooth and precise movements

The 10 Effects of Aging Changes

  1. People lose bone mass or density as they age, especially women after menopause. The bones lose calcium and other minerals.
  2. The spine is made up of bones called vertebrae. Between each bone is a gel-like cushion (called a disk). The middle of the body (trunk) becomes shorter as the disks gradually lose fluid and become thinner.
  3. Vertebrae also lose some of their mineral content, making each bone thinner. The spinal column becomes curved and compressed (packed together). Bone spurs caused by aging and overall use of the spine may also form on the vertebrae.
  4. The foot arches become less pronounced, contributing to a slight loss of height.
  5. The long bones of the arms and legs are more brittle because of mineral loss, but they do not change length. This makes the arms and legs look longer when compared with the shortened trunk.
  6. The joints become stiffer and less flexible. Fluid in the joints may decrease. The cartilage may begin to rub together and wear away. Minerals may deposit in and around some joints (calcification). This is common in the shoulder.
  7. Hip and knee joints may begin to lose cartilage (degenerative changes). The finger joints lose cartilage and the bones thicken slightly. Finger joint changes are more common in women. These changes may be inherited.
  8. Lean body mass decreases. This decrease is partly caused by a loss of muscle tissue (atrophy). The speed and amount of muscle changes seem to be caused by genes. Muscle changes often begin in the 20s in men and in the 40s in women.
  9. Lipofuscin (an age-related pigment) and fat are deposited in muscle tissue. The muscle fibers shrink. Muscle tissue is replaced more slowly. Lost muscle tissue may be replaced with tough fibrous tissue. This is most noticeable in the hands, which may look thin and bony.
  10. Muscles are less toned and less able to contract because of changes in the muscle tissue and normal aging changes in the nervous system. Muscles may become rigid with age and may lose tone, even with regular exercise.

Here are 5 tips to protect your joints

1. Stop smoking if you are a smoker!

Smoking and tobacco use are risk factors for everything from cardiovascular problems to cancer. Smoking can hamper your joints, too.

2. Replace energy drinks and soda with water

Water makes up about 80% of your body’s cartilage (the flexible, connective tissue that cushions your joints). If you don’t stay well-hydrated, your body will pull water from cartilage and other areas

3. Don’t let extra weights overtax your joints

Your joints are meant to sustain a certain amount of force. If you are overweight or underweight, you’re likely putting more stress on your joints. A hearty mix of fruits and vegetables, as well as whole grains and healthy fats, can help to reduce your inflammation and protect your heart.

4. Always warm-up and cool down

If you skip the warm-up and start your exercise will put your joints at greater risk of strain and overloading. For the best result, we recommend the warm-up and cool-down exercise should take at least five minutes. Work with the same muscles you will use during exercise, but at a slow pace. Warm-up exercise is most important as you age because older joints are often less resilient.

5. Taking joints supplement – Quan Wei Active Joint

quan wei active joint

Quan Wei Active Joint is made from a blend of minerals formula (Glucosamine Sulfate, Chondroitin, MSM) and herbs (Morinda, Epimedium, Sambucus, etc.). Glucosamine is commonly taken in combination with chondroitin to help patients suffering from joint problems, particularly those who suffer from osteoarthritis.

How will Quan Wei Active Joint benefit me?

  • Regenerates and repair cartilage cells
  • Recondition joint function
  • Support articular cartilage
  • Improve cartilage’s elasticity
  • Control the balance of the synovial fluid secretion
  • Enhances liver vitality
  • Combats poor calcium absorption

How to use: Take twice daily, 2 capsules each time.

Packing size: 90+30 capsules.

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Herbal Info

Butea Superba: The Benefits & Side Effects of Red Kwao Krua

What is Butea Superba (Red Kwao Krua)

Butea Superba (Red Kwao Krua) is an androgenic herb widely used among the males of Thailand as an aphrodisiac and to improve erectile quality.

Found in the hills of  Thailand, a natural compound is definitely creating a worldwide sexual sensation. It has a molecular structure that makes it a natural PDE 5 inhibitor making it a perfect natural male enhancer.

This plant grows in the open and the long roots of the plant are buried under the ground, similar to the roots of a yam. The roots of the mature plant are 8 to 9 inches long before they turn into tubers in the shape of elephant tusks. On cutting, the tubers reveal many red fibers and leak red sap. This type of plant reproduces through seeds and the separation of its roots.

The Health Benefit of Butea Superba

The majority of evidence to support Butea Superba’s potential health benefits comes from preliminary research on animals, although a few small clinical trials and case reports have been published.

Butea Superba as a Testosterone Booster

There is a case study where a 35-years old Thai man was diagnosed with hyperandrogenaemia after using an unreported dose of Red Kwao for “few weeks”. In fact, his lab tests showed his dihydrotestosterone (DHT) levels to be at 1512 pg/mL (reference values are between 250-990 pg/mL) and his principal “side-effect” was, as you can guess, a very high sex drive.

After closer inspection by medical experts, the “problematic” source of this increased androgenic (read: masculinity) was found to be Butea Superba, which the man said he had been taking to prevent hair loss. The man was then told to stop the usage of B. Superba, and one week after cessation of the herb, his DHT levels had returned back to normal and his sexual drive was also back to “normal”.

Sperm Count

Butea Superba has been studied in animal models as a potential fertility enhancer in men. A 2006 study of rats found eight weeks of Butea Superba treatment increased sperm counts by 16% compared to controls. However, there is no research linking the herb to increased sperm counts in humans and it is too soon to recommend it as a treatment for low sperm counts.

Butea Superba is used as a sex enhancer in Asia by middle-aged and older men as a tonic and virility enhancer.

Researchers and academics had found that Butea Superba products could be in both forms – ingest products such as a health food product and a topical application product such as a gel product. The delivery of Butea Superba can be through an oral ingestion capsule or extracts from this herb can be formulated into gel form for external application.

Possible Side Effects

Although little is known about the safety of regular use of Butea Superba, findings from animal-based research indicate that the herb may have adverse effects on blood chemistry and testosterone levels. Some research suggests it raises testosterone levels, however, additional studies suggest high doses of the herb may have the opposite effect.

Butea Superba is believed to act similarly to other hormones, including follicle-stimulating hormone (FSH), gonadotropic releasing hormone (GnRH), and testosterone. People who are undergoing hormone treatments or taking anabolic steroids should not take Butea Superba.

Butea Superba has been shown to increase androgen levels, which has been linked to polycystic ovary syndrome (PCOS), increased facial and body hair, and acne in women. Pregnant women should not take Butea Superba.

Given the potential health risks of this supplement, consult a physician prior to using Butea Superba is advised.

Where to buy Butea Superba Products?

There aren’t many vendors who sell Butea Superba, but I’ve found.

VITROMAN sells in both gel and capsule form.