Categories
Women's Health

Impotent and Female Orgasm

Although emphasis has been placed upon the role of premature ejaculation in the etiology of primary orgasmic dysfunction, primary or secondary impotence also contributes. Again the basic theme of man and woman coital interaction must be emphasized.

If there is not a sexually effective male partner, the female partner has the dual handicap of fear for her husband’s sexual performance as well as for her own.

If there is no penile erection there will be no effective coital connection.

Frequently women married to impotent men cannot accept the idea of developing a masturbatory facility or being manipulated to orgasm as a substitute for tension release.

However, if there has been a masturbatory pattern established before coital inadequacy assumes dominance, most women can return to this sexual outlet. In this situation, there is sufficient dominance of the previously conditioned biophysical structure to overcome negative input from a psychosocial system distressed by sexual performance fears.

But if there have been no previous substitute measures established, many women cannot turn to this mode of relief once impotence halts effective coital connection. In this situation, the psychosocial structure, unopposed by prior biophysical conditioning, assumes the dominant influence in the woman’s sexual response pattern.

Mr. and Mrs. D

were referred to the treatment of orgasmic inadequacy after four years of marriage. When seen in therapy she was 27 and her husband 43 years old. He had been married twice previously. There were children of both marriages and none in the current marriage.

The husband also was sexually dysfunctional in that he was secondarily impotent.

His second marriage had been terminated due to his inability to continue the effective coital connection. Although, when the unit was seen in consultation the marriage had been consummated, coitus occurred only once or twice a year.

Mrs. D’s background reflected somewhat limited financial means. Her father had died when the three siblings were young, and the family had been raised by their mother, who worked while the grandmother took care of the children.

Clothes were hand-me-downs, food the bare essentials. Her education had of necessity terminated with high school, and she worked as a receptionist in several different offices befo3e her marriage. She continued to live at home while working and contributed to what salary she made to help with the family’s limited income.

Mrs. D met her future husband when he visited the office where she worked. He invited the young woman to lunch. She accepted and married him four months later without knowledge of his sexual inadequacy, although she had been somewhat puzzled by his lack of forceful sexual approach during the brief courtship.

Mrs. D’s own sexual history had been one of a few unsuccessful attempts at masturbation, numerous petting episodes with boys in and out of high school, but no attempted coital connection. She had never been orgasmic.

Her husband had inherited a large estate and his financial situation certainly was the determining factor in his wife’s marital commitment. Since the girl had been distressed by a family background of genteel poverty, she felt the offer of marriage to be her real opportunity both to escape her environment and to help her two younger siblings.

The wedding trip was an unfortunate experience for Mrs. D when she realized for the first time that her husband had major functional difficulties. She knew little of male sexual response beyond the petting experiences but did try to help him achieve an erection by a multiplicity of stimulative approaches in his direction. There was no success in erective attainment.

The marriage was not consummated until six months after the ceremony. In the middle of the night, Mr. D awoke with an erection, moved to his wife, and inserted the penis. She experienced mild pain but reacted with pleasure, feeling that progress had been made.

However, following the usual pattern of .a secondarily impotent male, progress was fleeting. As stated, there were only a few other coital episodes in the course of the four-year marriage. In these instances, she always was awakened from sleep by her husband when he awoke to find himself with an erection.

Quick Ejaculation

Then there was rapid intromission and quick ejaculation. There was no history of a successful sexual approach by her husband under her conscious direction, insistence, or stimulation. Mr. D tried repetitively during long-continued manipulative and oral-genital sessions to bring his wife to orgasm, without result.

When they were referred for treatment, neither husband nor wife described sexual activity outside the marriage.

There have been 193 women treated for primary orgasmic dysfunction during the past 11years. Basically, in this method of therapy, the sexually dysfunctional woman is approached through her sexual value system.

If its requirements are non-serving limited, unrealistic, or inadequate to the marital relationship-by suggestion she is given an opportunity, with her husband’s help, to manipulate her biophysical and psychosocial structures of influence until an effective sexual value system is formed.

 
Categories
Women's Health

Sexual Lubrication

Sexual Lubrication

Probably the most frequent cause for dyspareunia originating with symptoms of burning, itching, or aching is a lack of adequate production of vaginal lubrication with sexual functioning.

During attempted penile intromission or with long-maintained coital connection, there must be adequate lubrication or there will be irritative distress for either or both coital partners.

Adequate production of vaginal lubrication is for women the physiological equivalent of erective attainment for men and her representation to her male partner of psychological readiness for vaginal penetration.

A consistent lack of functional levels of vaginal lubrication is as near as any woman can come to paralleling a man’s lack of effective erection.

Inadequate development of vaginal lubrication has many causes, but by far the most common is lack of interest in the particular opportunity or identification with the involved sexual partner. Women must experience positive input from their sexual value systems if they are to respond repetitively and effectively to their mate’s sexual approaches.

Women who cannot think or feel sexually, those reflecting fears of sexual performance, and even those acting out a spectator’s role in sexual functioning, either do not develop sufficient vaginal lubrication to support a painless coital episode or, if penetrated successfully, they cease production of the necessary lubricative material shortly after intravaginal penile thrusting is initiated.

Deep Penetration

Additionally, women fearful of pain with deep vaginal penetration, fearful of pregnancy with any coital connection, fearful of exposure to social compromise, and fearful of sexual inadequacy are frequently poor producers of vaginal lubrication. Any Component of fear present in sexual experiences reduces the receptivity to sensate input, thereby blunting biophysical responsivity.

If there is insufficient vaginal lubrication, there may or may not be acute pain with full penile intromission, depending primarily upon the parity of the woman, but they’re usually will be vaginal burning, irritation, or aching both during and after coital connection.

There are tens of thousands of women who become markedly apprehensive at the onset of any definitive sexual approach, simply because they know severe vaginal irritation will be experienced not only during but frequently for hours after any significantly maintained coital connection.

Not to be forgotten as a specific segment among the legions of women afflicted by inadequate production of vaginal lubrication are those in their postmenopausal years. If they are not supported by adequate sex-steroid-replacement techniques, the production of vaginal lubrication usually drops off markedly as the vaginal mucosa routinely turns atrophic.

There well may be aching and irritation in the vagina for a day or two after a coital connection for women contending with this evidence of sex-steroid starvation.

A major category of women tending to be poor lubrication producers during coital connection is that a significant segment of the female population with overt lesbian orientation. Many women psychosexually committed to a homophile orientation attempt regularity of coital connection for socioeconomic reasons. Frequently, they may not lubricate well during heterosexual activity, although there usually is ample lubrication when they are directly involved in the homosexual expression.

In most instances:

Inadequate production of vaginal lubrication can be reversed with a definite therapeutic approach. Certainly, women burdened by a multiplicity of sex-oriented fears can be provided psychotherapeutic relief of their phobias and subsequently reversed with relative ease from their particular pattern of sexual inadequacy.

Those women with chronic vaginal itching and irritation can be protected from continuing dyspareunia because both infectious and chemical vaginitis are reversible under proper clinical control. Senile vaginitis responds in short order to adequate sex-steroid-replacement techniques.

There are only two major categories of women for whom the co-therapists have little to offer to constitute effective production of vaginal lubrication: first, women mated to men for whom they have little or no personal identification, understanding, affection, or even sexual respect; and second, homosexually oriented women practicing coition for socioeconomic reasons with no interest in their male companions as sexual partners.

Undesired Sex

As opposed to the symptoms of aching, irritation, or burning in the vagina, complaints of severe pain developed during penile thrusting provide the most difficulty in delineating between subjective and objective etiology.

Although many women register this type of complaint when seeking to avoid undesired sexual approaches, there are some basic pathological conditions in the female pelvis that can and do engender severe pain in response to active coital connection.

One of the difficulties in delineating the severity of the complaint of dyspareunia is to identify pelvic pathology of a quality sufficient to support the female partner’s significant complaints of painful coition. The pelvic residual from severe infection or pelvic implants of endometriosis usually is easily identified by adequate pelvic and rectal examinations.

These clinical entities will be discussed briefly in context later in the topic. Current attention is drawn to probably the most frequently overlooked of the major physiological syndromes creating intense pelvic pain during coital connection.

Categories
Women's Health

Sex, Pelvic Syndromes

One of the most obscure of pelvic pathological syndromes, yet one of the most psychosexually crippling, is traumatic laceration of the ligaments supporting the uterus. This syndrome was described clinically. Five women have been referred to the Foundation for relief of subjective symptoms of dyspareunia after referral sources had assured the husbands that there was no plausible physical reason for the constant complaint of severe pain with deep penile thrust.

These 5 postpartum women had severe broad ligament lacerations and were relieved of their distress by definitive surgical approaches, not by psychotherapy. Therefore, they do not represent a component of the statistical analysis of treatment for sexual dysfunction.

Three women reflecting the onset of dyspareunia after criminal abortion techniques also have been seen in consultation and are not reflected in the statistics of the sexual inadequacy study. Three more women have been seen in gynecological consultation for acquired dyspareunia after gang-rape experiences.

They also have not been an integral part of the sexual-dysfunction study. These clinical problems will be mentioned in context. When first seen clinically, women with traumatic lacerations of the uterine supports, acquired with delivery or by specific criminal abortion techniques, present complaints that commonly accrue from pelvic vasocongestion, dyspareunia, dysmenorrhea, and a feeling of being excessively tired.

These complaints are secondary or acquired in nature. The traumatized women consistently can relate the onset of their acquired dyspareunia to one particular obstetrical experience even from among three or four such episodes.

The basic intercourse distress arises with deep penetration of the penis. Women describe the pain associated with intercourse to be as if their husbands had “hit something” with the penis during deep vaginal penetration.

These involved women may note other physical irritations frequently seen with chronic pelvic vasocongestion, a constantly nagging backache, throbbing or generalized pelvic aching, and occasionally, a sense that “everything is falling out.”

These symptoms are made worse in any situation requiring a woman to be on her feet for an exceptional length of time, as a full day spent doing heavy housecleaning or working as a saleswoman in a department store.

Most women lose interest in any regularity of sexual expression when distressed by acquired dyspareunia. Handicapped by constant anticipation of painful pelvic stimuli created by penile thrusting, they also may lose any previously established facility for orgasmic return.

The basic pathology of the syndrome of broad ligament laceration is confined to the soft tissues of the female pelvis. The striking features of the pelvic examination are the position of the uterus and almost always in severe third-degree retroversion and the particularly unique feeling that develops for the examiner with manipulation of the cervix.

This portion of the uterus feels just as if it were being rotated as a universal joint. It may be moved in any direction, up, down, laterally, or on an anterior-posterior plane with minimal, if any, correspondingly responsive movement of the corpus and body of the uterus.

Even the juncture of the cervix to the lower uterine segment is ill-defined. The feeling is one of an exaggerated Hegar’s sign of early pregnancy, in which the cervix appears to move in a manner completely independent of the attached corpus.

In addition to the “universal joint” feeling returned to the examiner when moving the cervix, a severe pain response usually is elicited by any type of cervical movement. However, pain is primarily occasioned by pushing the cervix in an upward plane.

In the more severe cases, either in advanced bilateral broad ligament laceration or in a presenting complaint of five years or more in duration even mild lateral motion of the cervix will occasion a painful response. During the examination, the retroverted uterus appears to be perhaps twice increased in size.

Pressing against the corpus in the cul-de-sac to reduce the third-degree retroversion also will produce a marked pain response. When the examiner applies upward pressure on the cervix or pressure in the cul-de-sac against the corpus, the patient frequently responds to the painful stimulus by stating, “It’s just like the pain I have with intercourse.”

A detailed obstetrical history is a salient feature in establishing the diagnosis of the broad ligament laceration syndrome. The untoward obstetrical event creating the lacerations may be classified as surgical obstetrics, as an obstetrical accident, or even as a poor obstetrical technique.

Occasionally, women with both the positive pelvic findings and the subjective symptoms of this syndrome cannot provide a positive history of obstetrical trauma, but this situation does not rule out the existence of the syndrome.

Many women are unaware of an unusual obstetrical event because they were under advanced degrees of sedation or even full anesthesia at the time.

Precipitate deliveries, difficult forceps deliveries, complicated breech deliveries, and postmature-infant deliveries are all suspect as obstetrical events that occasionally contribute to tears in the maternal soft parts.

If these tears are established in the supports of the uterus (broad ligaments) the immediate postpartum onset of severe dyspareunia can be explained anatomically and physiologically. It is important to emphasize clinically that although a woman may not be able to describe a specific obstetrical misfortune in her history, she well may be able to date the onset of her acquired dyspareunia to one particular obstetrical event.

Three cases have been seen in which a criminal abortion was performed by extensively packing the vaginal barrel, leading ultimately to dilation of the cervix and expulsion of uterine content.

These extensive vaginal-packing episodes created tears of the broad ligaments, completely parallel to those occasioned by actual obstetrical trauma. Each woman, although unaware of the etiological significance, dated the onset of the symptoms of acquired dyspareunia to the specific experience with the vaginal-packing type of abortive technique.

 
Categories
Overall Health

5 Tips to Protect Your Joints

What are the Joints?

joint or articulation is the connection made between bones in the body which link the skeletal system into a functional whole. They are constructed to allow for different degrees and types of movement. Some joints, such as the knee, elbow, and shoulder, are self-lubricating, almost frictionless, and are able to withstand compression and maintain heavy loads while still executing smooth and precise movements

The 10 Effects of Aging Changes

  1. People lose bone mass or density as they age, especially women after menopause. The bones lose calcium and other minerals.
  2. The spine is made up of bones called vertebrae. Between each bone is a gel-like cushion (called a disk). The middle of the body (trunk) becomes shorter as the disks gradually lose fluid and become thinner.
  3. Vertebrae also lose some of their mineral content, making each bone thinner. The spinal column becomes curved and compressed (packed together). Bone spurs caused by aging and overall use of the spine may also form on the vertebrae.
  4. The foot arches become less pronounced, contributing to a slight loss of height.
  5. The long bones of the arms and legs are more brittle because of mineral loss, but they do not change length. This makes the arms and legs look longer when compared with the shortened trunk.
  6. The joints become stiffer and less flexible. Fluid in the joints may decrease. The cartilage may begin to rub together and wear away. Minerals may deposit in and around some joints (calcification). This is common in the shoulder.
  7. Hip and knee joints may begin to lose cartilage (degenerative changes). The finger joints lose cartilage and the bones thicken slightly. Finger joint changes are more common in women. These changes may be inherited.
  8. Lean body mass decreases. This decrease is partly caused by a loss of muscle tissue (atrophy). The speed and amount of muscle changes seem to be caused by genes. Muscle changes often begin in the 20s in men and in the 40s in women.
  9. Lipofuscin (an age-related pigment) and fat are deposited in muscle tissue. The muscle fibers shrink. Muscle tissue is replaced more slowly. Lost muscle tissue may be replaced with tough fibrous tissue. This is most noticeable in the hands, which may look thin and bony.
  10. Muscles are less toned and less able to contract because of changes in the muscle tissue and normal aging changes in the nervous system. Muscles may become rigid with age and may lose tone, even with regular exercise.

Here are 5 tips to protect your joints

1. Stop smoking if you are a smoker!

Smoking and tobacco use are risk factors for everything from cardiovascular problems to cancer. Smoking can hamper your joints, too.

2. Replace energy drinks and soda with water

Water makes up about 80% of your body’s cartilage (the flexible, connective tissue that cushions your joints). If you don’t stay well-hydrated, your body will pull water from cartilage and other areas

3. Don’t let extra weights overtax your joints

Your joints are meant to sustain a certain amount of force. If you are overweight or underweight, you’re likely putting more stress on your joints. A hearty mix of fruits and vegetables, as well as whole grains and healthy fats, can help to reduce your inflammation and protect your heart.

4. Always warm-up and cool down

If you skip the warm-up and start your exercise will put your joints at greater risk of strain and overloading. For the best result, we recommend the warm-up and cool-down exercise should take at least five minutes. Work with the same muscles you will use during exercise, but at a slow pace. Warm-up exercise is most important as you age because older joints are often less resilient.

5. Taking joints supplement – Quan Wei Active Joint

quan wei active joint

Quan Wei Active Joint is made from a blend of minerals formula (Glucosamine Sulfate, Chondroitin, MSM) and herbs (Morinda, Epimedium, Sambucus, etc.). Glucosamine is commonly taken in combination with chondroitin to help patients suffering from joint problems, particularly those who suffer from osteoarthritis.

How will Quan Wei Active Joint benefit me?

  • Regenerates and repair cartilage cells
  • Recondition joint function
  • Support articular cartilage
  • Improve cartilage’s elasticity
  • Control the balance of the synovial fluid secretion
  • Enhances liver vitality
  • Combats poor calcium absorption

How to use: Take twice daily, 2 capsules each time.

Packing size: 90+30 capsules.

Categories
Herbal Info

Butea Superba: The Benefits & Side Effects of Red Kwao Krua

What is Butea Superba (Red Kwao Krua)

Butea Superba (Red Kwao Krua) is an androgenic herb widely used among the males of Thailand as an aphrodisiac and to improve erectile quality.

Found in the hills of  Thailand, a natural compound is definitely creating a worldwide sexual sensation. It has a molecular structure that makes it a natural PDE 5 inhibitor making it a perfect natural male enhancer.

This plant grows in the open and the long roots of the plant are buried under the ground, similar to the roots of a yam. The roots of the mature plant are 8 to 9 inches long before they turn into tubers in the shape of elephant tusks. On cutting, the tubers reveal many red fibers and leak red sap. This type of plant reproduces through seeds and the separation of its roots.

The Health Benefit of Butea Superba

The majority of evidence to support Butea Superba’s potential health benefits comes from preliminary research on animals, although a few small clinical trials and case reports have been published.

Butea Superba as a Testosterone Booster

There is a case study where a 35-years old Thai man was diagnosed with hyperandrogenaemia after using an unreported dose of Red Kwao for “few weeks”. In fact, his lab tests showed his dihydrotestosterone (DHT) levels to be at 1512 pg/mL (reference values are between 250-990 pg/mL) and his principal “side-effect” was, as you can guess, a very high sex drive.

After closer inspection by medical experts, the “problematic” source of this increased androgenic (read: masculinity) was found to be Butea Superba, which the man said he had been taking to prevent hair loss. The man was then told to stop the usage of B. Superba, and one week after cessation of the herb, his DHT levels had returned back to normal and his sexual drive was also back to “normal”.

Sperm Count

Butea Superba has been studied in animal models as a potential fertility enhancer in men. A 2006 study of rats found eight weeks of Butea Superba treatment increased sperm counts by 16% compared to controls. However, there is no research linking the herb to increased sperm counts in humans and it is too soon to recommend it as a treatment for low sperm counts.

Butea Superba is used as a sex enhancer in Asia by middle-aged and older men as a tonic and virility enhancer.

Researchers and academics had found that Butea Superba products could be in both forms – ingest products such as a health food product and a topical application product such as a gel product. The delivery of Butea Superba can be through an oral ingestion capsule or extracts from this herb can be formulated into gel form for external application.

Possible Side Effects

Although little is known about the safety of regular use of Butea Superba, findings from animal-based research indicate that the herb may have adverse effects on blood chemistry and testosterone levels. Some research suggests it raises testosterone levels, however, additional studies suggest high doses of the herb may have the opposite effect.

Butea Superba is believed to act similarly to other hormones, including follicle-stimulating hormone (FSH), gonadotropic releasing hormone (GnRH), and testosterone. People who are undergoing hormone treatments or taking anabolic steroids should not take Butea Superba.

Butea Superba has been shown to increase androgen levels, which has been linked to polycystic ovary syndrome (PCOS), increased facial and body hair, and acne in women. Pregnant women should not take Butea Superba.

Given the potential health risks of this supplement, consult a physician prior to using Butea Superba is advised.

Where to buy Butea Superba Products?

There aren’t many vendors who sell Butea Superba, but I’ve found.

VITROMAN sells in both gel and capsule form.

Categories
Herbal Info

Catuaba – Improved Sexual Health for Men

There is a popular expression in Brazil

“If the father is 60 and below, the son is his; after 60years old, the son belongs to Catuaba.”

No, Catuaba is not a fertility god, Catuaba is actually a small, flowering tree that’s native to the Amazon. Among the trees used for Catuaba (a tribal word meaning “what gives strength to the Indian”) are Erythroxylum caatingae, Trichilia catigua, Anemopaegma arvense, and Micropholis caudata. Hundreds of years ago, Brazil’s native Tupi tribe discovered that Catuaba bark has aphrodisiac qualities.  Drinking Catuaba tea to spawn erotic dreams and boost libido became a part of their culture.

Now, Catuaba is one of the most popular Amazonian aphrodisiac plants in the world and is included in many male enhancement formulas.

How Does Catuaba Bark Enhance Sexual Health?

Within Brazilian herbal medicine, Catuaba bark is categorized as a stimulant and is even related to the coca plant. But, you can relax. Catuaba doesn’t contain any of the alkaloids found in cocaine. Catuaba bark does contain, however, three specific alkaloids believed to support a healthy libido. Some Catuaba even contains yohimbine, another natural aphrodisiac.

Research involving animal models has shown that the Catuaba bark may enhance erectile strength by widening blood vessels, allowing more blood to flow to the penis. Catuaba may even have some neurological benefits due to its antioxidant content. It’s been observed to increase the brain’s sensitivity to dopamine, which makes sex more pleasurable.

Supplementing with Catuaba Bark. A downswing in sexual energy can happen for a number of reasons: a lack of physical fitness, medications, and the age-related symptoms of andropause.

Catuaba bark has been used by many men across the world to rejuvenate their libido and desires and is not associated with adverse health effects. Oddly enough, while some herbal aphrodisiacs are gender-specific, women too may experience the aphrodisiac benefits of Catuaba bark.

VITROMAN BRAZILIAN CATUABA

catuaba, brazilian catuaba, catuaba bark

Vitroman Brazilian Catuaba contains a Brazilian herb that is known as an herbal supplement deriving from a small tree native to the Brazilian landscape. It has yellow and orange flowers and bears an oval-shaped, yellowish-brown fruit. Its bark is well known for its uncommon antiviral and antibacterial qualities.

Brazilian herbalists believe that the composition or color of a fruit or herb, or the color of its extract, indicates the organ upon which it operates — the organ to be cured or remedied. The extract of Catuaba bark is red, which links it to the blood, liver, and circulatory system.

Effect:

  • Help achieves erection & increase desire.
  • Regains lost sexual function.
  • Stimulates central nervous, boosts energy level.
  • Control pain & fights fatigue.
  • Anti-depression, anti-anxiety, improve good mood.
  • Provides energy & immune support.

You can purchase from here -> Vitroman.com

Categories
Men's Health Overall Health Women's Health

Testosterone: What it is

Testosterone is a hormone behind muscle-building, fat-burning, libido, and even strongly affects mood and energy.

The testicles are the main source of testosterone production in men while the ovaries are in charge of producing this sex hormone in women. However, in women, levels of testosterone are typically lower compared to men. However, abnormally low testosterone levels in women (as well as men) can contribute to symptoms and may indicate an underlying health issue.

In general, men begin to experience an increase in testosterone production during puberty, with testosterone levels gradually declining to start at about age 30. When natural testosterone levels begin to lower, both men and women can experience a number of different symptoms.

Low testosterone levels

Low testosterone levels in men can lead to symptoms that can affect many different aspects of health and well-being. Many men that experience a decrease in testosterone report sleep disturbances and insomnia, emotional changes such as depression, and issues related to their sexual performance/desires. Along with these symptoms, some men even face changes in fertility, decreased strength, and weight gain.

Athletic performance can also suffer due to loss of energy, as well as increased difficulty building muscle and burning fat. Having greater body fat and less muscle can then potentially increase the risk of heart disease, diabetes, and other conditions dependent on optimal metabolism.

Low levels of testosterone, also called low T levels, can produce a variety of symptoms in men, including:

  • decreased sex drive
  • less energy
  • weight gain
  • feelings of depression
  • moodiness
  • low self-esteem
  • less body hair
  • thinner bones

How to boosts your testosterone?

Boosting your testosterone level with oyster extract is just what you need in order to help with problems like a low sex drive, no energy, or impotence. Essentially, the oyster extract is the powdered up dried meat of an oyster. It is made into a tablet or put into a capsule to make it ready for consumption. Oyster extract is also often used by men and athletes in order to help the body boost testosterone naturally due to the high levels of zinc it is made up of.

Zinc is a mineral that lots of men are not getting enough of daily even though it plays an important role in the creation of testosterone in your system. Oyster extract is also an extremely rich source of vitamin D too. Vitamin D is another nutrient that increasing numbers of are not getting enough of regularly since it is not typically found in food.

Vitamin D also helps your body create more testosterone, so it can help you improve your muscles and increase libido as well.

Oysters are an aphrodisiac, meaning they can help enhance libido and sexual performance, mainly in men. The zinc found in the oyster extract is incredible, it is made up of more zinc per serving compared to any other food.

Zinc has been associated with sexual problems in men. In fact, erectile dysfunction can be a sign of zinc deficiency. As a result, eating oysters can provide men with the zinc necessary to increase their libido and perform well.

Vitroman Oyster Ext offers benefits from oyster meat useful to support men’s health. Oyster provides a natural source of multi-minerals and marine vitamins such as amino acids, taurine, and zinc. It plays important role in enhancing metabolism and energy-boosting. Regular intake aid in physical fitness and vigor.

Oyster Extract helps increase fertility, boost sperm count. Low Sperm count affects many men who wish to have children. Oyster extract carries a spermatogenesis compound which can increase its activity. It is rich in protein (peptides) and Zinc naturally that stimulates the production of testosterone thereby raising its levels in the body.

Categories
Senior Health

Ejaculations, Seminal Fluid

Seminal-fluid volume is gradually reduced during the aging process.

In the younger man with 24-36 hours of prior ejaculatory continence, the total, seminal-fluid volume averages 3-5 ml, while with a similar continence pattern, an output of 2-3 ml is within normal limits for the post 50 male.

These definitive physiological changes seem not to detract from the aging male’s orgasmic experience, subjective interpretation of which usually is one of extreme sensate pleasure.

The orgasmic episode is fully enjoyed, regardless of whether the first stage is altered significantly or even totally missing from the experience.

Obvious reductions in ejaculatory pressure and volume do not alter the male’s basic focus upon the sensate pleasure of the experience. The clinical concern that develops with the advent of these physical changes in the cycle of sexual response occurs when aging males do not understand the physiological appropriateness of their altered sexual response patterning.

If a man who experiences a brief one stage orgasmic episode and ejaculates a reduced seminal fluid volume under little or no pressure does not understand that these altered reaction patterns are naturally occurring phenomena after voluntarily prolonged excitement or plateau phases of sexual tension, he may become extremely concerned about his sexual functioning.c

He may be frightened by the fallacious concept that he is in the process of losing his ability to function in a sexually effective manner.

The fact

On the very next occasion for a coital connection, there may be very rapid progress from excitement through the plateau to a two-stage orgasmic process, significant ejaculatory pressure, and an adequate seminal-fluid volume does not appease the anxious male.

He has noted specific physiological variants in aging sexual functioning on at least one occasion and is aware of no logical explanation for their development.

It never occurs to him that during the first episode, when there was the marked alteration of his usual response pattern, the marital partners were selectively directing themselves to the wife’s pleasure, while during the second experience the sexual partners had turned the tables and obviously were intent upon deriving male release and sexual satiation.

Following the usual dictates of our culture, when any alteration occurs in the structuring of man’s sexual response pattern that he does not understand, he falls into the psychosocial trap of the cultural demand for the constancy of male sexual performance and worries about the possible loss of masculinity.

The resolution phase of the older man’s sexual response cycle also evidences marked physiological alteration from his previously established response patterning. As the male ages, his refractory period, the period following ejaculation, during which the male is biophysically unresponsive to sexual stimuli, extends in a parallel fashion.

The refractory period of the younger man usually continues for but a matter of minutes before he can return to full erection under the influence of effective sexual stimulation.

The refractory period for the aging male occasionally may continue for a matter of minutes, but usually, it is a matter of hours before return to full erection is possible.

Again, if this phenomenon is understood by women as well as by men, the older man will not worry about being unable to respond to a repetitive mounting opportunity as he could when in the 20 to 40 year age group.

Neither he nor his wife will be creating fears for sexual performance if there is no attempt to force erective return when he is in a physiologically extended refractory period. It also should be pointed out that, as opposed to the younger man;

The aging male may lose his erection after ejaculation quickly.

There may not be a two-stage loss of erection as in the younger man’s natural response pattern.

Frequently, the older man’s penis returns to its flaccid state in a matter of seconds after ejaculation, instead of the younger man’s pattern of minutes or even hours.

The informed older man will not be concerned by his response variants if educated to understand that the variants are natural results of physiological involution. But should he not have this information, the penis’s literally falling from the vagina immediately after ejaculation can stimulate real fears for the adequacy of performance.

When an uninformed older man endures the first experience of losing an’ erection so rapidly, he immediately may wonder whether he will be able to achieve a fully effective erection the next time there is a coital opportunity.

When he worries about erective capacity, he tends to try to force or will an effective erection with subsequent coital exposure. Then he is in difficulty.

A plea must be entered for the wide dissemination of information on the natural physiological variants of the aging male’s sexual response cycle, to support not only the men but also the women in our society.

The wife of the 50 to 70-year-old man also must understand the natural evolutionary changes inherent in her husband’s aging process. Once she appreciates the continuing male facility for sexual expression regardless of changed response pattern, she will be infinitely more comfortable about importuning her husband sexually.

She will not worry about his delayed erection time when fully aware that it does not mean that he no longer finds her attractive. The less than fully erect penis sometimes present in the plateau phase can be readily inserted by a perceptive woman with the sure knowledge after successful intromission that her husband’s first few penile strokes will aid in the full development of the erection.

An informed wife will not hesitate to be sexually demonstrative when she realizes that once a coital connection has been established her husband has increased facility for ejaculatory control.

Confident of her own and her husband’s facility to respond successfully, even though the typical response patterns of their younger years have been altered, the concerned wife can meet her husband freely without the usual cultural reservations.

This security of sexual performance for the aging man and woman comes only from the wide dissemination of information from authoritative sources.

Categories
Senior Health

ED in Aging Male

Composite case studies have been selected to identify and illustrate the dysfunctional characteristics of the male aging process.

Both Mr. and Mrs. A were 66 and 62 years of age when referred to the Foundation for sexual inadequacy. They had been married 39 years and had three children, the youngest of whom was 23 years of age. All children were married and living outside the home.

They had maintained reasonably effective sexual interchange during their marriage.

Mr. A had no difficulty with erection, reasonable ejaculatory control, and, aside from two occasions of prostitute exposure, had been fully committed to the marriage. Mrs. A occasionally orgasmic during intercourse and regularly orgasmic during her occasional masturbatory experiences had continued regularity of coital exposure with her husband until five years before referral for therapy.

Mr. A had recently retired from a major manufacturing concern. He had been relatively successful in his work and there were no specific financial problems facing man and wife during their declining years.

Both members of the marital unit had enjoyed good health throughout the marriage. At age 61, he had taken his wife abroad on a vacation trip which entailed many sightseeing trips with a different city on the agenda almost every day.

They were chronically tired during the exhausting trip, but because they were on vacation and away from home there was a definite increase over the established frequency of coital connections. Mr. A noted for the first time slowed erective attainment.

Regardless of his level of sexual interest or the depth of his wife’s commitment to the specific sexual experience, it took him progressively longer to attain a full erection. With each sexual exposure his concern for the delay in erective security increased until finally, just before termination of the vacation trip, he failed for the first time to achieve an erection quality sufficient for vaginal penetration.

When the coital opportunity first developed after return home, erection was attained, but again it was quite slow in development. The next two opportunities were only partially successful from an erective point of view, and thereafter he was secondarily impotent.

After several months they consulted their physician and were assured that this loss of erective power comes to all men as they age and that there was nothing to be done. Loath to accept the verdict, they tried on several occasions to force an erection with no success. Mr. A was seriously depressed for several months but recovered without apparent incident.

Approximately 18 months after the vacation trip, the couple had accepted their “fate.” The impotence was acknowledged to be a natural result of the aging process. This resigned attitude lasted approximately four years.

Although initially the marital unit and their physician had fallen into the socio-cultural trap of accepting the concept of sexual inadequacy as an aging phenomenon, the more Mr. and Mrs. A considered their dysfunction the less willing they were to accept the blanket concept that lack of erective security was purely the result of the aging process.

They reasoned that they were in good health, had no basic concerns as a marital unit, and took good care of themselves physically. Therefore, why was this dysfunction to be expected simply because some of their friends reportedly had accepted the loss of male erective prowess as a natural occurrence?

Each partner underwent a thorough medical checkup and sought several authoritative opinions, refusing to accept the concept of the irreversibility of their sexual distress. Finally, approximately five years after the onset of a full degree of secondary impotence, they were referred for treatment.

Sexual functioning was reconstituted for this marital unit within the first week after they arrived at the Foundation and as soon as they could absorb and accept the basic material directed toward the variation in the physiological functioning of the aging male.

No longer were they concerned with the delay in erective attainment; there were no more attempts to will, force, or strain to accomplish erection under assumed pressures of performance.

In short:
They needed only the security of the knowledge that the response pattern which initially had raised the basic fear of dysfunction was a perfectly natural result of the involutional process.

When they could accept the fact that it naturally took longer for an older man to achieve an erection, particularly if he were tired or distracted, the basis for their own sexual inadequacy disappeared.

Some six years after termination of the acute phase of therapy, this couple, now in the early seventies and late sixties, continue coital connection once or twice a week.

The husband has learned to ejaculate on his own demand schedule, and neither partner attempts a rapid return to sexual function after a mutually satisfactory sexual episode.

Husband and wife B

The husband, age 62, and his wife, age 63, were referred to the Foundation. They had two children, both of whom were married and lived out of the home.

Their sexual dysfunction had begun when the husband was 57 years old. He had noted some delay in attaining erection and marked reduction in ejaculatory volume and was particularly concerned with the fact that the ejaculatory experience was one of the mere dribbling of seminal fluid from the external urethral meatus, under obviously reduced pressure.

All these involutional signs and symptoms developed within approximately a year after he had noticed some delay in onset of erection attainment.

The more he worried about his symptoms the more frequent the occasions of impotence.

Mrs. B was completely convinced that this pattern of sexual involution was true to be expected as part of the aging process.

Rather than distress her husband, she suggested that they use separate bedrooms.

She changed from a pattern of free and easy exchange of sexual demand to one of availability for coital connection only at her husband’s expression of interest. To resolve her own sexual tensions, she masturbated about once every ten days to two weeks without her husband’s knowledge.

Finally, Mr. B developed severe prostatic spasm with ejaculation during approximately half the increasingly rare occasions when there was sufficient erective security to establish a coital connection. It was the persistence of this symptom of pain that first brought medical consultation and ultimately referral for treatment.

In the evaluation of this man during the physical examination, there was marked muscular weakness noted, a history of easy fatigability, and increasing lassitude in physical expression.

Mr. B also had been distressed in the last two to three years before referral to therapy with distinct memory loss for recent events. He described the loss of work effectiveness for approximately the same length of time.

With these overt symptoms suggestive of steroid starvation, testosterone replacement was initiated empirically. Within those days there was a partial return of ejaculatory pressure and a moderate shortening of the time span for the delayed erective reaction. The prostatic pain did not recur.

Husband and wife B Steroid Replacement

Once Mrs. B could accept the explanation for the onset of her husband’s sexual dysfunction, she was pleased to return to the role of an active sexual partner. The coital connection has continued regularly for the past three years with both members of the marital unit supported by steroid-replacement techniques.

In brief, for the sexually dysfunctional aging male, the primary concern is one of education so that both the man and his wife can understand the natural involutional changes that can develop within their established pattern of sexual performance.

Sex steroid replacement should be employed only if definite physical evidence of the male climacteric exists. As the newer techniques for establishing testosterone levels in blood serum become more widely disseminated, it will be infinitely easier to define and describe the male climacteric and therefore to offer testosterone replacement to those who need it, on a more definitive basis than an empirical diagnosis.

With effective dissemination of information by proper authority, the aging man can be expected to continue in a sexually effective manner into his ninth decade. Fears of performance are engendered by a lack of knowledge of the natural involutional changes in male sexual responsivity that accompany the aging process.

Really, the only factor that the aging male must understand is that loss of erective prowess is not a natural component of aging.

Statistical evaluation of the aging population and a consideration of treatment failure rates will constitute the following section. This material arbitrarily has been placed in the male rather than the female.

First, it was felt important to keep the statistical consideration of the aging marital units together, and second, because there were 56 males So years old or over in marital units accepted for treatment and only 37 wives 50 or older, it seemed appropriate to include the brief statistical discussion in the chapter reflecting the larger segment of the aging population.

 
Categories
Senior Health

Aging Male Sex

Aging Male Sex

The natural aging process creates some specific physiological changes in the male cycle of sexual response. Knowledge of these cycle variations has not been widely disseminated.

There have been little concept of a physiological basis for differentiating between natural sexual involution and pathological dysfunction when considering the problems of male sexual dysfunction in the post-so age group.

If all too few professionals are conversant with anticipated alterations in male sexual functioning created by the aging process, how can the general public be expected to adjust to the internal alarms raised by these naturally occurring phenomena?

Tragically, yet understandably, tens of thousands of men have moved from effective sexual functioning to varying levels of secondary impotence as they age, because they did not understand the natural variants that physiological aging imposes on previously established patterns of sexual functioning.

Sexually Impaired at 50

From a psychosexual point of view, the male over age 50 has to contend with one of the great fallacies of our culture. Every man in this age group is arbitrarily identified by both public and professional alike as sexually impaired.

When the aging male is faced with unexplained yet natural involutional sexual changes and deflated by widespread psychosocial acceptance of the fallacy of sexual incompetence as a natural component of the aging process, is it any wonder that he carries a constantly increasing burden of fear of performance?

Before discussing specifics of sexual dysfunction in the aging population, the natural variants that the aging process imposes on the established male cycle of sexual response should be considered.

For sake of discussion, the four phases of the sexual response cycle excitement, plateau, orgasm, and resolution will be employed to establish a descriptive framework. Also for descriptive purposes, the term older man will be used about the male population from 50 to 70 years of age, and the term younger man used to describe the 20 to 40 year age group.

In recent years the younger man’s sexual response cycle has been established with physiological validity and will serve as a baseline for comparison with the physiological variations of aging.

If an older man can be objective about his reactions to sexual stimuli during the excitement phase, he may note a significant delay in erective attainment compared to his facility of response as a younger man.

Most older men do not establish an erective response to effective sexual stimulation for a matter of minutes, as opposed to a matter of 9f seconds as younger men, and the erection may not be as full or as demanding as that to which previously he has been accustomed.

It simply takes the older man longer to be fully involved subjectively in acceptance and expression of any form of sensate stimulation.

If natural delays in reaction time are appreciated, there will be no panic on the part of either husband or wife. If, however, the aging male is uninformed and not anticipating delayed physiological reactions to sexual stimuli, he may indeed panic and responding in the worst possible way to try to will or force an erection.

The unfortunate results of this approach to erective security have been discussed at length in the treatment of impotence.

Aging Male Erections

As the aging male approaches the plateau phase, his erection usually has been established with fair security. There may be little if any testicular elevation, a negligible amount of scrotal-sac vasocongestion, and minimal deep vascular engorgement of the testes.

Most older men who have had a pre-ejaculatory fluid emission (Cowper’s gland secretory activity) will notice either total absence of, or marked reduction in, the amount of this pre-ejaculatory emission as they age.

From the aspect of time-span, the plateau phase usually lasts longer for an older man than for his younger counterpart. When an aging male reaches that level of elevated sexual tension identified as thoroughly enjoyable, he usually can and frequently does wish to maintain this plateau-phase level of sensual pleasure for an indefinite period of time without becoming enmeshed by ejaculatory demand.

This response pattern is age-related; the younger man tends to drive for early ejaculatory release when plateau-phase levels of sexual tension have accrued. One of the advantages of the aging process with specific reference to sexual functioning is that.

Generally speaking, control of ejaculatory demand in the 50 to 70 year age group is far better than in the 20 to 40 year age group.

In the cycle of sexual response, the largest number of physiological changes to come within an objective focus for older men occurs during the orgasmic phase (ejaculatory process). The orgasmic phase is relatively standardized for younger men, varying minimally in duration and intensity of experience unless influenced by the psychosexual opposites of long-continued continence or a high level of sexual satiation.

For younger men, the entire ejaculatory process is divided into two well-recognized stages. The first stage, ejaculatory inevitability, is a brief period of time (2 to 4 seconds) during which the male feels the ejaculation coming and no longer can control it before ejaculation actually occurs.

These subjective symptoms of ejaculatory inevitability are created physiologically by regularly recurring contractions of the prostate gland and, questionably, the seminal vesicles. Contractions of the prostate begin at o.8-second intervals and continue through both stages of the male orgasmic experience.

The second stage of the orgasmic phenomenon consists of the expulsion of the seminal-fluid bolus accrued under pressure in the membranous and prostatic portions of the urethra, through the full length of the penile urethra.

Again, there are regularly recurring 0.8-second inter-contractile intervals. This specific interval lengthens after the first three or four contractions of the penile urethra in younger men.

Subjectively, the sensation is one of the flows of a volume of warm fluid under pressure and emission of the seminal fluid bolus in ejaculatory spurts with a pressure sufficient to expel fluid content distances of 12 to 24 inches beyond the urethral meatus.

As the male ages, he develops many individual variants on the basic theme of the two-stage orgasmic experience described for the younger man. Usually, his orgasmic experience encompasses a shorter time span.

There may not be even a recognizable first stage to the ejaculatory experience so that an orgasmic experience without the stage of ejaculatory inevitability is quite a common occurrence.

Even with a recognizable first stage, there still may be marked variation in reaction patterns. Occasionally, the older man’s phase of ejaculatory inevitability lasts but a second or two as opposed to the younger man’s pattern ranging from 2 to 4 seconds.

In an older man’s first-stage experience, there may be only one or two contractions of the prostate before involuntary initiation of the second stage, seminal-fluid expulsion.

Alternatively, the first stage of orgasmic experience may be held for as long as 5 to 7 seconds. Occasionally the prostate, instead of contracting within the regularly described pattern of 0.8-second intervals, develops a spastic contraction, creating subjectively the sense of ejaculatory inevitability.

Inadequate Testosterone

The prostate may not relax from spasm into rhythmically expulsive contractions for several seconds, hence the 5-7-second duration of the first-stage experience. In addition to objective variants in a first-stage orgasmic episode, there may be no possible objective or subjective definition of the first stage of orgasmic experience at all.

The stage of ejaculatory inevitability may be totally missing from the aging male’s sexual response cycle. A single-stage orgasmic episode develops clinically in two circumstances.

The first circumstance is that of clinical dysfunction developing as the result of inadequate testosterone production.

Actually, the lack of a recognizable first stage in orgasmic experience can result from a low sex-steroid level for the male just as steroid starvation in the female may produce an orgasmic experience of markedly brief duration.

The second occasion of an absent first stage in the orgasmic experience develops after there has been a prior denial of ejaculatory opportunity over a long period of intravaginal containment to satisfy the aging male’s coital partner sexually.

There also are obvious physiological changes in the second stage of the orgasmic experience that develop with the aging process.

The expulsive contractions of the penile urethra have onset at 0.8second intervals but are maintained for only one or two contractions at this rate:

The expulsive force delivering the seminal fluid bolus externally, so characteristic of second-stage penile contractions in the younger man, also is diminished, with the distance of unencumbered seminal-fluid expulsion ranging from 3 to 12 inches from the urethral meatus.